Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Normal responses of atrial natriuretic factor and renal tubular function to sodium loading in hypertension-prone humans.

Blood pressure - 1 Jan 2000

Bruun N E, Dige-Pedersen H, Skøtt P

Abstract excerpt

BACKGROUND: In order to explore the hypothesis of an atrial natriuretic factor (ANF) deficiency in prehypertension, we compared the response to sodium loading on ANF and renal function in subjects with positive and negative histories of hypertension. METHODS: Twenty-two offspring of hypertensive parents (OH) and 20 offspring of normotensive parents (ON) were studied after 4 days of low (50 mmol/day) or high (300...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Atrial Natriuretic Factor
Share to Cyclic GMP
Share to Genetic Predisposition to Disease

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Glomerular Filtration Rate

Share to Hypertension

Share to Kidney Tubules

Share to Sodium, Dietary

Source

Identifiers

PMID: 11055473
DOI: 10.1080/080370500439092

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Normal responses of atrial natriuretic factor and renal tubular function to sodium loading in hypertension-prone humans.PMID 11055473Read ↗

Sodium and kidney disease.Semantic proximity Read ↗

Collecting Duct Nitric Oxide Synthase 1ß Activation Maintains Sodium Homeostasis During High Sodium Intake Through Suppression of Aldosterone and Renal Angiotensin II Pathways.Semantic proximity Read ↗

Salt-sensitive blood pressure--an intermediate phenotype predisposing to diabetic nephropathy?Semantic proximity Read ↗

Neuronal nitric oxide synthase-deficient mice have impaired renin release but normal blood pressure.Semantic proximity Read ↗

Early alteration in glomerular reserve in humans at genetic risk of essential hypertension: mechanisms and consequences.Semantic proximity Read ↗

Increasing evidence for the role of salt and salt-sensitivity in hypertension.Semantic proximity Read ↗

Blood pressure responses to dietary sodium: Association with autonomic cardiovascular function in normotensive adults.Semantic proximity Read ↗

Dietary salt excess unmasks blunted aldosterone suppression and sodium retention in the stroke-prone phenotype of the spontaneously hypertensive rat.Semantic proximity Read ↗

Contribution of circulating angiotensinogen concentrations to variations in aldosterone and blood pressure in a group of African ancestry depends on salt intake.Semantic proximity Read ↗

Renal arterial dysfunction, impaired pressure natriuresis and salt-sensitivity in a mouse model of Cushing syndromeSemantic proximity Read ↗

Lanosterol Synthase Gene Polymorphisms and Changes in Endogenous Ouabain in the Response to Low Sodium Intake.Semantic proximity Read ↗

Sodium intake and cardiovascular health.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.