Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Alterations in spontaneous single unit activity of striatal subdivisions during ontogenesis in mutant dystonic hamsters.

Brain research - 13 Mar 1999

Gernert M, Richter A, Löscher W

Abstract excerpt

The pathophysiology of idiopathic dystonia, characterized by sustained twisting movements and postures, is still unknown. Clinically, however, the basal ganglia are thought to be the main causative origin of idiopathic dystonia. In the dtsz hamster, a genetic animal model for idiopathic paroxysmal dystonia, the attacks occur in response to mild stress and the severity of dystonia is age-dependent. Previous...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Cricetinae
Share to Dystonia
Share to Electrophysiology

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Share to gamma-Aminobutyric Acid

Share to Mesocricetus

Share to Motor Neurons

Share to Movement Disorders

Share to Neostriatum

Source

Identifiers

PMID: 10064814
DOI: 10.1016/s0006-8993(99)01080-x

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Alterations in spontaneous single unit activity of striatal subdivisions during ontogenesis in mutant dystonic hamsters.PMID 10064814Read ↗

Spontaneous remission of paroxysmal dystonia coincides with normalization of entopeduncular activity in dt(SZ) mutants.Semantic proximity Read ↗

[3H]-2-deoxyglucose uptake study in mutant dystonic hamsters: abnormalities in discrete brain regions of the motor system.Semantic proximity Read ↗

Deficit of striatal parvalbumin-reactive GABAergic interneurons and decreased basal ganglia output in a genetic rodent model of idiopathic paroxysmal dystonia.Semantic proximity Read ↗

Regional alterations in neuronal activity in dystonic hamster brain determined by quantitative cytochrome oxidase histochemistry.Semantic proximity Read ↗

Quantitative EEG analysis of depth electrode recordings from several brain regions of mutant hamsters with paroxysmal dystonia discloses frequency changes in the basal ganglia.Semantic proximity Read ↗

Polyamines in a genetic animal model of paroxysmal dyskinesia.Semantic proximity Read ↗

Developmental profile of the aberrant dopamine D2 receptor response in striatal cholinergic interneurons in DYT1 dystonia.Semantic proximity Read ↗

Impairment of bidirectional synaptic plasticity in the striatum of a mouse model of DYT1 dystonia: role of endogenous acetylcholine.Semantic proximity Read ↗

Altered postnatal maturation of striatal GABAergic interneurons in a phenotypic animal model of dystonia.Semantic proximity Read ↗

Effects of intrastriatal injections of glutamate receptor antagonists on the severity of paroxysmal dystonia in the dtsz mutant.Semantic proximity Read ↗

Role of striatal NMDA receptor subunits in a model of paroxysmal dystonia.Semantic proximity Read ↗

Changes in AMPA receptor binding in an animal model of inborn paroxysmal dystonia.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.