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Article

iNOS is not responsible for nitrosylation of RyR1 in mdx mice with truncated dystrophin

2020-07-07

Abstract excerpt

<title>Abstract</title> <p><bold>Background: </bold>Previous research indicated that nitric oxide synthase (NOS) is the key molecule for S-nitrosylation of ryanodine receptor 1 (RyR1) in DMD model mice (<italic>mdx</italic> mice) and that both neuronal NOS (nNOS) and inducible NOS (iNOS) might contribute to the reaction because nNOS is mislocalized in the cytoplasm and iNOS expression is higher in <italic>mdx</it...

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Literature Corpus work
deddf556-a1fa-56c8-b6db-8c5a3a0e6677
DOI
10.21203/rs.3.rs-16865/v2
Open publication

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