Back to search

Article

A Novel Secretome Rewrites the Immune Response in Viral Acute Respiratory Distress Syndrome

2026-02-09

Abstract excerpt

<h4>Background</h4> Hyperinflammatory syndromes such as viral acute respiratory distress syndrome (ARDS) demand immunotherapies that are safe and effective at suppressing cytokine storm. As a novel treatment for ARDS, PRS CK STORM is proposed as a next-generation cell-free secretome derived from co-cultures of M2 macrophages and mesenchymal stromal cells. <h4>Methods</h4> We performed a double-blind, randomized...

Topics

Open a Topic to create a Post that cites this publication.

Identifiers and source

Literature Corpus work
80b40513-5d5d-5efb-b05e-a558d3317d52
DOI
10.64898/2026.02.06.704035
Open publication

Related research

Semantic proximity does not establish scientific evidence.

Click a neighbor to travelStep 1 · 12 closest
Interactive article relationship graphSelect a related publication card to move it into the centre and load its closest explainable connections. Solid lines are source-backed structured connections. Dashed lines are semantic discovery signals and are not scientific evidence.
A Novel Secretome Rewrites the Immune Response in Viral Acute Respiratory Distress SyndromeDOI 10.64898/2026.02.06.704035
Single-cell RNA sequencing reveals characteristics of myeloid cells in pulmonary post-acute sequelae of SARS-CoV-2Semantic proximity
Tracking inflammation resolution signatures in lungs after SARS-CoV-2 omicron BA.1 infection of K18-hACE2 miceSemantic proximity
Distinct Pulmonary Innate and Adaptive Immune Responses during the Initiation and Resolution of Natural and Breakthrough SARS-CoV-2 InfectionSemantic proximity
Peritoneal M2 macrophage-derived extracellular vesicles as natural multi-target nanotherapeutics to attenuate cytokine storm after severe infectionsSemantic proximity
Investigation of Lung-Blood Interplays in COVID-19 Cytokine Storm Using Gene Co-Expression and Protein-Protein Interaction NetworksSemantic proximity
ACE2 is the critical in vivo receptor for SARS-CoV-2 in a novel COVID-19 mouse model with TNF- and IFNγ-driven immunopathologySemantic proximity
Multilevel proteomics reveals host perturbations by SARS-CoV-2 and SARS-CoVSemantic proximity
ACE2 is the critical <i>in vivo</i> receptor for SARS-CoV-2 in a novel COVID-19 mouse model with TNF- and IFNγ-driven immunopathologySemantic proximity
COVID-19 and its Therapeutics: Special Emphasis on Mesenchymal Stem Cells Based TherapySemantic proximity
Surface proteins of SARS-CoV-2 drive airway epithelial cells to induce interferon-dependent inflammationSemantic proximity
SARS-CoV-2-Triggered Mast Cell Rapid Degranulation Induces Alveolar Epithelial Inflammation and Lung InjurySemantic proximity
Alveolitis in severe SARS-CoV-2 pneumonia is driven by self-sustaining circuits between infected alveolar macrophages and T cellsSemantic proximity
Select a neighboring publication to make it the new centre.