Expression of hypothalamic peptides in mice lacking neuronal nitric oxide synthase: reduced beta-END immunoreactivity in the arcuate nucleus.

doi:10.1159/000054390

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Article

Expression of hypothalamic peptides in mice lacking neuronal nitric oxide synthase: reduced beta-END immunoreactivity in the arcuate nucleus.

Neuroendocrinology - 1 Dec 1998

Bernstein H G, Keilhoff G, Seidel B, Stanarius A, Huang P L, Fishman M C, Reiser M, Bogerts B, Wolf G

Abstract excerpt

The gas nitric oxide (NO) is an important messenger in brain signaling. Along with many other functions, NO is thought to influence the expression and/or release of various hypothalamic hormones (corticotropin-releasing hormone (CRH), gonadotropin-releasing hormone (GnRH) and vasopressin). To lea...

Topics

Animals
Arcuate Nucleus of Hypothalamus
Hypothalamus
Immunohistochemistry
In Situ Hybridization
Mice
Mice, Knockout
Mutation
Nerve Tissue Proteins
Neuropeptides
Nitric Oxide Synthase
Nitric Oxide Synthase Type I
Pro-Opiomelanocortin
RNA, Messenger
beta-Endorphin

Source

Open in PubMed

Identifiers

PMID: 9873204
DOI: 10.1159/000054390

Genes

POMC
CRH

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 30 Aug 2026. Prepared record publication-9873204-2026-08-30.1. Manifest publication-graph-cohort-2026-08-30.1.

Source receipt: PubMed via Helena Literature Corpus, retrieved 30 Aug 2026.

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