Article
Thyroid disease mediated by molecular defects in cell surface and nuclear receptors.
The American journal of medicine - 1 Dec 1998
Bodenner D L, Lash R W
Abstract excerpt
The proposed mechanisms of RTH are not mutually exclusive. In fact, there is considerable experimental evidence that many if not all of these complex receptor interactions with elements of the transcriptional unit are involved in RTH. Several aspects of RTH remain unclear, in particular on a clin...
Topics
- Autoantibodies
- GTP-Binding Proteins
- Humans
- Mutation
- Receptors, Cell Surface
- Receptors, Cytoplasmic and Nuclear
- Receptors, Thyrotropin
- Thyroid Diseases
- Thyroid Hormones
