Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Hypertensive response to chronic NO synthase inhibition is different in Sprague-Dawley rats from two suppliers.

The American journal of physiology - 1 Nov 1998

Pollock D M, Rekito A

Abstract excerpt

Experiments were conducted to determine whether Sprague-Dawley rats from different suppliers have the same hypertensive response to chronic inhibition of nitric oxide synthase. Rats (240-260 g) obtained from either Harlan or Charles River Laboratories were maintained in metabolic cages for baseline (week 0) measurements before receiving Nomega-nitro-L-arginine methyl ester (L-NAME) in the drinking water for 2 wk...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Enzyme Inhibitors
Share to Genetic Variation
Share to Hypertension

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Share to Nitric Oxide Synthase

Share to Rats, Sprague-Dawley

Share to Species Specificity

Source

Identifiers

PMID: 9791095
DOI: 10.1152/ajpregu.1998.275.5.R1719

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Hypertensive response to chronic NO synthase inhibition is different in Sprague-Dawley rats from two suppliers.PMID 9791095Read ↗

Predisposition of spontaneously hypertensive rats to develop renal injury during nitric oxide synthase inhibition.Semantic proximity Read ↗

Renal baroreceptor-stimulated renin in the eNOS knockout mouse.Semantic proximity Read ↗

Neuronal nitric oxide synthase-deficient mice have impaired renin release but normal blood pressure.Semantic proximity Read ↗

Renal cortical and medullary blood flow responses to L-NAME and ANG II in wild-type, nNOS null mutant, and eNOS null mutant mice.Semantic proximity Read ↗

Impact of Renal Denervation on Circadian Variations of Blood Pressure and Clock Gene Expression in Spontaneously Hypertensive RatsSemantic proximity Read ↗

Role of alpha(2)-adrenergic receptor subtypes in the acute hypertensive response to hypertonic saline infusion in anephric mice.Semantic proximity Read ↗

Expression of nitric oxide synthase isoforms in hypothalamo-pituitary-adrenal axis during the development of spontaneous hypertension in rats.Semantic proximity Read ↗

Neuronal nitric oxide synthase activity in the paraventricular nucleus buffers central endothelin-1- induced pressor response and vasopressin secretion.Semantic proximity Read ↗

Role of nNOS in blood pressure regulation in eNOS null mutant mice.Semantic proximity Read ↗

Androgen-receptor defect abolishes sex differences in nitric oxide and reactivity to vasopressin in rat aorta.Semantic proximity Read ↗

Early blockade of bradykinin B2-receptors alters the adult cardiovascular phenotype in rats.Semantic proximity Read ↗

Inducible nitric oxide synthase (iNOS) is not required for IL-2-induced hypotension and vascular leak syndrome in mice.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.