Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Alterations in the Expression of Chemokine Mrna Levels in Fibrosis-Resistant and -Sensitive Mice After Thoracic Irradiation

1 Jan 1998

Abstract excerpt

Fibrosis, characterized by the accumulation of collagen, is a consequence of a chronic inflammatory response. The purpose of this study was to determine if the mRNA expression of the chemokines, lymphotactin (Ltn), RANTES, eotaxin, macrophage inflammatory protein (MIP)-1 alpha, -1 beta, and -2, interferon-inducible protein 10 (IP-10), and monocyte chemotactic protein-1 (MCP-1), are altered during the development...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Effects of Radiation Exposure
Share to Interstitial Lung Diseases and Idiopathic Pulmonary Fibrosis
Share to NF-κB Signaling Pathways

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 9635254
DOI: 10.3109/01902149809041538

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 5 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Alterations in the Expression of Chemokine Mrna Levels in Fibrosis-Resistant and -Sensitive Mice After Thoracic IrradiationPMID 9635254Read ↗

The Th1/Th17 balance dictates the fibrosis response in murine radiation-induced lung disease.Semantic proximity Read ↗

Radiation induced pulmonary fibrosis as a model of progressive fibrosis: Contributions of DNA damage, inflammatory response and cellular senescence genesSemantic proximity Read ↗

Mechanisms of the alternative activation of macrophages and non-coding RNAs in the development of radiation-induced lung fibrosisSemantic proximity Read ↗

Early alterations in extracellular matrix and transforming growth factor β gene expression in mouse lung indicative of late radiation fibrosisSemantic proximity Read ↗

Pathophysiological mechanisms underlying phenotypic differences in pulmonary radioresponseSemantic proximity Read ↗

Role of Infiltrating Monocytes in the Development of Radiation-Induced Pulmonary Fibrosis.Semantic proximity Read ↗

Combined CXCR1/CXCR2 antagonism decreases radiation-induced alveolitis in the mouse.Semantic proximity Read ↗

Radiation-induced lung fibrosis in a tumor-bearing mouse model is associated with enhanced Type-2 immunity.Semantic proximity Read ↗

Global analysis of gene expression in pulmonary fibrosis reveals distinct programs regulating lung inflammation and fibrosisSemantic proximity Read ↗

Pathogenetic mechanisms in radiation fibrosis.Semantic proximity Read ↗

Inhibition of neutrophil degranulation by Nexinhib20 delays the development of radiation-induced pulmonary fibrosisSemantic proximity Read ↗

Modeling radiation-induced lung injury: lessons learned from whole thorax irradiationSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.