Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

In vivo protein binding and functional analysis of cis-acting elements in the U3 region of the bovine leukemia virus long terminal repeat.

Journal of virology - 1 Jul 1998

Xiao J, Buehring G C

Abstract excerpt

Bovine leukemia virus (BLV) is a member of the human T-cell leukemia virus (HTLV)/BLV group of retroviruses. These viruses regulate their own transcription by producing Tax, a protein which activates the virus promoter region, the long terminal repeat (LTR). To explore the molecular mechanisms involved in the transactivation, we identified protein binding elements by in vivo footprinting and analyzed their...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Binding Sites
Share to Cattle
Share to Cell Line

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Share to Mutation

Share to Promoter Regions, Genetic

Share to Protein Binding

Share to Repetitive Sequences, Nucleic Acid

Share to TATA Box

Source

Open in PubMed Open full text

Identifiers

PMID: 9621062
DOI: 10.1128/JVI.72.7.5994-6003.1998
PMCID: PMC110404

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

In vivo protein binding and functional analysis of cis-acting elements in the U3 region of the bovine leukemia virus long terminal repeat.PMID 9621062Read ↗

The region between amino acids 245 and 265 of the bovine leukemia virus (BLV) tax protein restricts transactivation not only via the BLV enhancer but also via other retrovirus enhancers.Semantic proximity Read ↗

Transcription factor AP-4 participates in activation of bovine leukemia virus long terminal repeat by p34 Tax.Semantic proximity Read ↗

Mutant tax protein from bovine leukemia virus with enhanced ability to activate the expression of c-fos.Semantic proximity Read ↗

Identification of bovine leukemia virus tax function associated with host cell transcription, signaling, stress response and immune response pathway by microarray-based gene expression analysis.Semantic proximity Read ↗

Construction and functional characterization of mutants of the bovine leukaemia virus trans-activator protein p34tax.Semantic proximity Read ↗

Regulation of Expression and Latency in BLV and HTLV.Semantic proximity Read ↗

A mutant form of the tax protein of bovine leukemia virus (BLV), with enhanced transactivation activity, increases expression and propagation of BLV in vitro but not in vivo.Semantic proximity Read ↗

RNA polymerase III control elements are required for trans-activation by the murine retroviral long terminal repeat sequences.Semantic proximity Read ↗

Deacetylase inhibitors and the viral transactivator TaxBLV synergistically activate bovine leukemia virus gene expression via a cAMP-responsive element- and cAMP-responsive element-binding protein-dependent mechanism.Semantic proximity Read ↗

Protein and DNA elements involved in transactivation of the promoter of the bovine herpesvirus (BHV) 1 IE-1 transcription unit by the BHV alpha gene trans-inducing factorSemantic proximity Read ↗

Identification of novel factors which bind specifically to the core promoter of the immunoglobulin heavy chain gene.Semantic proximity Read ↗

Identification of functional domains within the bICP0 protein encoded by bovine herpesvirus 1.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.