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Article

[The AAP and PS1/2 mutations linked to early onset familial Alzheimer's disease increase the extracellular concentration and A beta 1-42 (43)].

Rinsho shinkeigaku = Clinical neurology - 1 Dec 1997

Younkin S G

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Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

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PMID: 9577655

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PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

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[The AAP and PS1/2 mutations linked to early onset familial Alzheimer's disease increase the extracellular concentration and A beta 1-42 (43)].PMID 9577655Read ↗

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Presenilins and Alzheimer's disease: the role of A beta 42.Semantic proximity Read ↗

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Secreted amyloid beta-protein similar to that in the senile plaques of Alzheimer's disease is increased in vivo by the presenilin 1 and 2 and APP mutations linked to familial Alzheimer's disease.Semantic proximity Read ↗

A New Pathogenic Mutation in the APP Gene (I716V) Increases the Relative Proportion of A 42(43)Semantic proximity Read ↗

Familial Alzheimer's disease-linked presenilin 1 variants elevate Abeta1-42/1-40 ratio in vitro and in vivo.Semantic proximity Read ↗

[Dominant forms of Alzheimer's disease: from genotype to phenotype].Semantic proximity Read ↗

Physiochemical characterization of the Alzheimer's disease-related peptides A beta 1-42Arctic and A beta 1-42wt.Semantic proximity Read ↗

Synthesis, aggregation, neurotoxicity, and secondary structure of various Aβ1–42 mutants of familial Alzheimer's disease at positions 21–23Semantic proximity Read ↗

The beta APP717 Alzheimer mutation increases the percentage of plasma amyloid-beta protein ending at A beta 42(43)Semantic proximity Read ↗

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