Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Glial fibrillary acidic protein as a marker of perisinusoidal stellate cells that can distinguish between the normal and myofibroblast-like phenotypes.

Biology of the cell - 1 Jan 1996

Buniatian G, Hamprecht B, Gebhardt R

Abstract excerpt

Glial fibrillary acidic protein (GFAP) has recently been shown to provide a marker for normal perisinusoidal stellate cells (PSC) in the liver. However, nothing was known so far about the changes in the intercellular abundancy of GFAP during transformation of PSC into myofibroblasts, a process characterized by marked changes in the expression of elements of the cytoskeleton. In order to address this question, we...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Biomarkers
Share to Cell Nucleus
Share to Cells, Cultured

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Share to Fibroblasts

Share to Phenotype

Share to Transformation, Genetic

Source

Identifiers

PMID: 9004488

Genes

GFAP

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Glial fibrillary acidic protein as a marker of perisinusoidal stellate cells that can distinguish between the normal and myofibroblast-like phenotypes.PMID 9004488Read ↗

Acquisition of blood--tissue barrier--supporting features by hepatic stellate cells and astrocytes of myofibroblastic phenotype. Inverse dynamics of metallothionein and glial fibrillary acidic protein expression.Semantic proximity Read ↗

The myofibroblastic conversion of peribiliary fibrogenic cells distinct from hepatic stellate cells is stimulated by platelet-derived growth factor during liver fibrogenesis.Semantic proximity Read ↗

Phenotypic change in portal fibroblasts in biliary fibrosis.Semantic proximity Read ↗

Fibrogenic cell phenotype modifications during remodelling of normal and pathological human liver in cultured slices.Semantic proximity Read ↗

Clinical significance and gene expression study of human hepatic stellate cells in HBV related-hepatocellular carcinoma.Semantic proximity Read ↗

Smoothelin, a new marker to determine the origin of liver fibrogenic cells.Semantic proximity Read ↗

Intermediate filaments modulation in an in vitro model of the hepatic stellate cell activation or conversion into the lipocyte phenotype.Semantic proximity Read ↗

Long-lasting coexpression of nestin and glial fibrillary acidic protein in primary cultures of astroglial cells with a major participation of nestin(+)/GFAP(-) cells in cell proliferation.Semantic proximity Read ↗

GFAP-deficient astrocytes are capable of stellation in vitro when cocultured with neurons and exhibit a reduced amount of intermediate filaments and an increased cell saturation density.Semantic proximity Read ↗

Phenotypic modulation in lipocytes in experimental liver fibrosis.Semantic proximity Read ↗

The stellate cells phenotypic transformation in the CCl4- injured liver fibrosis of ICR mice: their desmin immunoreactivity and vitamin A storage.Semantic proximity Read ↗

[Effect of RAR-beta transfection on the proliferation and phenotype of rat hepatic stellate cells].Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.