Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Oncogene- transformed NIH 3T3 cells display radiation resistance levels indicative of a signal transduction pathway leading to the radiation-resistant phenotype.

Radiation research - 1 Aug 1993

Pirollo K F, Tong Y A, Villegas Z, Chen Y, Chang E H

Abstract excerpt

Oncogenes and their normal counterparts, proto-oncogenes, are functionally important cellular genes which interact with one another as components of signal transduction pathways leading to cell growth and differentiation. Numerous reports in the literature have also begun to link these genes to the phenomenon of cellular radiation resistance. In this report we examine the radiation resistance level of NIH 3T3...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to 3T3 Cells
Share to Cell Line, Transformed

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Share to Cell Survival

Share to Nuclear Proteins

Share to Phenotype

Share to Protein Serine-Threonine Kinases

Share to Signal Transduction

Source

Identifiers

PMID: 8367595

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Oncogene- transformed NIH 3T3 cells display radiation resistance levels indicative of a signal transduction pathway leading to the radiation-resistant phenotype.PMID 8367595Read ↗

Oncogenic basis of radiation resistance.Semantic proximity Read ↗

Evidence supporting a signal transduction pathway leading to the radiation-resistant phenotype in human tumor cells.Semantic proximity Read ↗

Induced proto-oncogene expression increases susceptibility to radiation-induced carcinogenesis.Semantic proximity Read ↗

Radiation-induced cell transformation: transformation efficiencies of different types of ionizing radiation and molecular changes in radiation transformants and tumor cell lines.Semantic proximity Read ↗

Effects of c-raf-1 and c-myc expression on radiation response in an in vitro model of human small-cell-lung carcinoma.Semantic proximity Read ↗

Multistep nature of X-ray-induced neoplastic transformation in mammalian cells: genetic alterations and instability.Semantic proximity Read ↗

Direct evidence for the contribution of activated N-ras and K-ras oncogenes to increased intrinsic radiation resistance in human tumor cell lines.Semantic proximity Read ↗

Targeting tumor cells by enhancing radiation sensitivity.Semantic proximity Read ↗

Oncogenes and radiation carcinogenesis.Semantic proximity Read ↗

Radiation-induced EGFR-signaling and control of DNA-damage repairSemantic proximity Read ↗

Transfection of rat embryo cells with mutant p53 increases the intrinsic radiation resistance.Semantic proximity Read ↗

Irradiation induces diverse changes in invasive potential in cancer cell linesSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.