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Article

Role of retinoblastoma gene product in p53-mediated DNA damage response.

Experimental cell research - 1 Dec 1994

Smith M L, Zhan Q, Bae I, Fornace A J

Abstract excerpt

The cellular response to DNA-damaging agents involves the activation of cell cycle checkpoints. Checkpoints provide a transient delay in cell cycle progression, presumably to allow time for the cell to repair the damage. A most important checkpoint, active in the G1 phase of the cell cycle, is mediated by the p53 tumor suppressor gene product. To investigate the role of downstream components of the cell cycle...

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Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

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Identifiers

PMID: 7982477
DOI: 10.1006/excr.1994.1356

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PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

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Role of retinoblastoma gene product in p53-mediated DNA damage response.PMID 7982477Read ↗

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