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Article

Increased opsonization of a prtH-defective mutant of Porphyromonas gingivalis W83 is caused by reduced degradation of complement-derived opsonins.

Journal of immunology (Baltimore, Md. : 1950) - 15 May 1995

Schenkein H A, Fletcher H M, Bodnar M, Macrina F L

Abstract excerpt

Periodontitis is a disease of the supporting structures of the teeth that is caused by bacteria whose common ecologic niche is the gingival crevice or the periodontal pocket. Tissue destruction occurs in spite of both local and systemic immune responses against such bacteria. Porphyromonas gingivalis is considered to be an important pathogen in some forms of human periodontitis and is particularly interesting...

Topics

Antigens, Bacterial
Bacterial Proteins
Complement C3
Cysteine Endopeptidases
Humans
Mutation
Neutrophils
Opsonin Proteins
Porphyromonas gingivalis

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

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Identifiers

PMID: 7730636

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 29 Aug 2026. Prepared record publication-7730636-2026-08-29.1. Manifest publication-graph-cohort-2026-08-29.1.

Source receipt: PubMed via Helena Literature Corpus, retrieved 29 Aug 2026.

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Increased opsonization of a prtH-defective mutant of Porphyromonas gingivalis W83 is caused by reduced degradation of complement-derived opsonins.PMID 7730636Read ↗

Virulence of a Porphyromonas gingivalis W83 mutant defective in the prtH gene.Semantic proximity Read ↗

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Fimbrial Proteins of <i>Porphyromonas gingivalis</i> Mediate In Vivo Virulence and Exploit TLR2 and Complement Receptor 3 to Persist in MacrophagesSemantic proximity Read ↗

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Altered expression and modification of proteases from an avirulent mutant of Porphyromonas gingivalis W50 (W50/BE1).Semantic proximity Read ↗

Mechanisms of Resistance of<i>Porphyromonas gingivalis</i>to Killing by Serum ComplementSemantic proximity Read ↗

The capsule of Porphyromonas gingivalis reduces the immune response of human gingival fibroblastsSemantic proximity Read ↗

Relevance of IgG receptor IIIb (CD16) polymorphism to handling of Porphyromonas gingivalis: implications for the pathogenesis of adult periodontitis.Semantic proximity Read ↗

Role of RgpA, RgpB, and Kgp proteinases in virulence of Porphyromonas gingivalis W50 in a murine lesion model.Semantic proximity Read ↗

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The vimE gene downstream of vimA is independently expressed and is involved in modulating proteolytic activity in Porphyromonas gingivalis W83.Semantic proximity Read ↗

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