Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Mendelian randomization studies in atopic dermatitis: causal insights across omics layers.

Frontiers in immunology - 1 Jan 2026

Chera Alexandra, Bucur Octavian, Bumbăcea Roxana-Silvia

Abstract excerpt

Atopic dermatitis (AD) is a chronic inflammatory skin disease, shaped by genetic, immune and environmental factors. Even though this complex interaction has been thoroughly studied, uncovering causal relationships between specific exposures and AD remains challenging. Mendelian randomization (MR) has emerged as a powerful tool for establishing causal inferences between exposures and outcomes, using genome-wide...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Dermatitis, Atopic
Share to Genetic Predisposition to Disease

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Genome-Wide Association Study

Share to Genomics

Share to Mendelian Randomization Analysis

Share to Multiomics

Share to Precision Medicine

Share to Proteomics

Source

Open in PubMed Open full text

Identifiers

PMID: 41836422
DOI: 10.3389/fimmu.2026.1717812
PMCID: PMC12979485

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Mendelian randomization studies in atopic dermatitis: causal insights across omics layers.PMID 41836422Read ↗

Mendelian Randomization Studies in Atopic Dermatitis: A Systematic Review.Semantic proximity Read ↗

Phenome-wide Mendelian randomization analysis reveals new risk factors for 4 inflammatory skin diseases.Semantic proximity Read ↗

Exploring the causal relationship between inflammatory cytokines and C-reactive protein levels and atopic dermatitis: a two-way two-sample Mendelian randomization studySemantic proximity Read ↗

Causal effects of circulating lipids and lipid-lowering drugs on the risk of atopic dermatitis: a mendelian randomization study.Semantic proximity Read ↗

Single-cell transcriptomic mendelian randomization and co-localization reveal immune-mediated regulatory mechanisms and drug targets in atopic dermatitis.Semantic proximity Read ↗

Mendelian randomization indicates that atopic dermatitis contributes to the occurrence of diabetesSemantic proximity Read ↗

Genetic association of lipid and lipid-lowering drug target genes with atopic dermatitis: a drug target Mendelian randomization study.Semantic proximity Read ↗

Integrating Mendelian randomization and multiple-trait colocalization to uncover cell-specific inflammatory drivers of autoimmune and atopic diseaseSemantic proximity Read ↗

The causal relationship between immune cells and atopic dermatitis: A bidirectional Mendelian randomization study.Semantic proximity Read ↗

Mendelian Randomization Analysis Reveals a Complex Genetic Interplay among Atopic Dermatitis, Asthma, and Gastroesophageal Reflux Disease.Semantic proximity Read ↗

Genetic Correlations and Causal Relationships Among Allergic Diseases: A Comprehensive Mendelian Randomization Study With Multiomic Mediation Analysis.Semantic proximity Read ↗

Investigating causal relationships between Body Mass Index and risk of atopic dermatitis: a Mendelian randomization analysisSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.