DDX3X mutation and Epstein-Barr virus cooperate to induce R-loop-dependent oncogenesis

Abstract excerpt

tumors. These findings provide a better understanding of the functional interaction of the DDX3X mutation with EBV to provoke R-loop-dependent oncogenesis, shedding light on the pathogenic mechanism of EBV and future therapeutic approaches targeting R-loops in diseases involving RNA helicase alterations.

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