Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Bioinformatic analysis predicts the regulatory function of noncoding SNPs associated with Long COVID-19 syndrome.

Immunogenetics - 1 Dec 2024

Maiti Amit K

Abstract excerpt

Long or Post COVID-19 is a condition of collected symptoms persisted after recovery from COVID-19. Host genetic factors play a crucial role in developing Long COVID-19, and GWAS studies identified several SNPs/genes in various ethnic populations. In African-American population two SNPS, rs10999901 (C>T, p = 3.6E-08, OR = 1.39, MAF-0,27, GRCH38, chr10:71584799 bp) and rs1868001 (G>A, p = 6.7E-09, OR = 1.40,...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Black or African American

Share to Computational Biology

Share to Genetic Predisposition to Disease

Share to Genome-Wide Association Study

Share to Hispanic or Latino

Share to interferon and immune responses

Share to Long-Term Effects of COVID-19

Share to Polymorphism, Single Nucleotide

Source

Identifiers

PMID: 39042286
DOI: 10.1007/s00251-024-01348-6

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 3 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Bioinformatic analysis predicts the regulatory function of noncoding SNPs associated with Long COVID-19 syndrome.PMID 39042286Read ↗

Unraveling Risk Genes of COVID-19 by Multi-Omics Integrative AnalysesSemantic proximity Read ↗

Regulatory Noncoding and Predicted Pathogenic Coding Variants of CCR5 Predispose to Severe COVID-19.Semantic proximity Read ↗

Integrative Multi-Omics Framework for Causal Gene Discovery in Long COVIDSemantic proximity Read ↗

A genome-wide association study for survival from a multi-centre European study identified variants associated with COVID-19 risk of deathSemantic proximity Read ↗

Integrative Genome-Wide Association Studies of COVID-19 Susceptibility and Hospitalization Reveal Risk Loci for Long COVIDSemantic proximity Read ↗

Altered and allele-specific open chromatin landscape reveals epigenetic and genetic regulators of innate immunity in COVID-19Semantic proximity Read ↗

Genome-wide Association Study of Long COVIDSemantic proximity Read ↗

Hypotheses and facts for genetic factors related to severe COVID-19Semantic proximity Read ↗

COVID-19 genetic risk variants are associated with expression of multiple genes in diverse immune cell types.Semantic proximity Read ↗

A catalog of associations between rare coding variants and COVID-19 outcomesSemantic proximity Read ↗

Multi-Layered Genomic and Clinical Analysis Identifies Novel Variants, Co-Occurring Single Nucleotide Polymorphism Pairs, and Clinical Determinants of Host-Pathogen Interaction in COVID-19 Severity.Semantic proximity Read ↗

Whole genome sequence meta-analyses reveal common and rare genetic associations with critical COVID-19Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.