Macrophage Ontogeny, Phenotype, and Function in Ischemia Reperfusion-Induced Injury and Repair.

doi:10.34067/KID.0000000000000376

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Article

Macrophage Ontogeny, Phenotype, and Function in Ischemia Reperfusion-Induced Injury and Repair.

Kidney360 - 1 Mar 2024

Maryam Bibi, Smith Morgan E, Miller Sarah J, Natarajan Hariharasudan, Zimmerman Kurt A

Abstract excerpt

AKI is characterized by a sudden, and usually reversible, decline in kidney function. In mice, ischemia-reperfusion injury (IRI) is commonly used to model the pathophysiologic features of clinical AKI. Macrophages are a unifying feature of IRI as they regulate both the initial injury response as well as the long-term outcome following resolution of injury. Initially, macrophages in the kidney take on a...

Topics

Mice
Animals
Macrophages
Cytokines
Phenotype
Tumor Necrosis Factor-alpha
Acute Kidney Injury
Renal Insufficiency, Chronic
Reperfusion Injury
Reperfusion
Ischemia

Source

Open in PubMed Open full text

Identifiers

PMID: 38297436
DOI: 10.34067/KID.0000000000000376
PMCID: PMC11000738

Genes

CCL2
TNF

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 30 Aug 2026.

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