Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Characterization of metabolic phenotypes and distinctive genes in mice with low-weight gain.

FASEB journal : official publication of the Federation of American Societies for Experimental Biology - 1 Jan 2024

Nemoto Shino, Kubota Tetsuya, Ishikura Tomoyuki, Nakayama Manabu, Kobayashi Atsuo, Yazaki Junshi, Uchida Kazuyo, Matsuda Masashi, Kondo Takashi, Ohara Osamu, Koseki Haruhiko, Koyasu Shigeo, Ohno Hiroshi

Abstract excerpt

Being overweight exacerbates various metabolic diseases, necessitating the identification of target molecules for obesity control. In the current study, we investigated common physiological features related to metabolism in mice with low weight gain: (1) G protein-coupled receptor, family C, group 5, member B-knockout; (2) gastric inhibitory polypeptide receptor-knockout; and (3) Iroquois-related...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Adipose Tissue, Brown
Share to Adipose Tissue, White

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Diet, High-Fat

Share to Energy Metabolism

Share to Mice, Inbred C57BL

Share to Mice, Knockout

Share to Obesity

Share to Weight Gain

Source

Identifiers

PMID: 38069905
DOI: 10.1096/fj.202301565R

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Characterization of metabolic phenotypes and distinctive genes in mice with low-weight gain.PMID 38069905Read ↗

The metabolic response to a high-fat diet reveals obesity-prone and -resistant phenotypes in mice with distinct mRNA-seq transcriptome profiles.Semantic proximity Read ↗

Slc7a8 Deletion Is Protective against Diet-Induced Obesity and Attenuates Lipid Accumulation in Multiple OrgansSemantic proximity Read ↗

Extensive weight loss reveals distinct gene expression changes in human subcutaneous and visceral adipose tissueSemantic proximity Read ↗

Comparative Transcriptomic Analysis of Obesity and Lipodystrophy Reveals Shared Mechanisms and Novel Targets in Adipose Tissue DysfunctionSemantic proximity Read ↗

Transcriptome analysis of the adipose tissue in a mouse model of metabolic syndrome identifies gene signatures related to disease pathogenesisSemantic proximity Read ↗

BRD7 deficiency leads to the development of obesity and hyperglycemia.Semantic proximity Read ↗

Dietary intervention improves health metrics and life expectancy of the genetically obese Titan mouse.Semantic proximity Read ↗

Changes in gene expression foreshadow diet-induced obesity in genetically identical mice.Semantic proximity Read ↗

<i>Slc7a8</i> deletion is protective against diet-induced obesity and attenuates lipid accumulation in multiple organsSemantic proximity Read ↗

Loss of CTRP10 results in female obesity with preserved metabolic healthSemantic proximity Read ↗

Translational potential of mouse models of human metabolic diseaseSemantic proximity Read ↗

Novel hyperplastic expansion of white adipose tissue underlies the metabolically healthy obese phenotype of male LFABP null miceSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.