Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Circulating monocytes expressing senescence‐associated features are enriched in COVID ‐19 patients with severe disease

15 Nov 2023

Abstract excerpt

Accurate biomarkers for predicting COVID-19 severity have remained an unmet need due to an incomplete understanding of virus pathogenesis and heterogeneity among patients. Cellular senescence and its pro-inflammatory phenotype are suggested to be a consequence of SARS-CoV-2 infection and potentially drive infection-dependent pathological sequelae. Senescence-associated markers in infected individuals have been...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Neutrophil, Myeloperoxidase and Oxidative Mechanisms
Share to Single-cell and spatial transcriptomics
Share to Telomeres, Telomerase, and Senescence

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 37969056
DOI: 10.1111/acel.14011

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 8 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Circulating monocytes expressing senescence‐associated features are enriched in COVID ‐19 patients with severe diseasePMID 37969056Read ↗

Cellular senescence as a potential mediator of COVID‐19 severity in the elderlySemantic proximity Read ↗

Longitudinal analyses reveal age-specific immune correlates of COVID-19 severitySemantic proximity Read ↗

Patients with moderate to severe COVID-19 have an impaired cytokine response with an exhausted and senescent immune phenotype.Semantic proximity Read ↗

A human circulating immune cell landscape in aging and COVID-19Semantic proximity Read ↗

Persistent immune dysregulation and metabolic alterations following SARS-CoV-2 infectionSemantic proximity Read ↗

Immunosenescence in atherosclerosis: A role for chronic viral infectionsSemantic proximity Read ↗

SARS-CoV-2 infects lung epithelial cells and induces senescence and an inflammatory response in patients with severe COVID-19Semantic proximity Read ↗

Clinical and molecular characteristics of COVID-19 patients with persistent SARS-CoV-2 infectionSemantic proximity Read ↗

SARS-CoV-2 infection triggers paracrine senescence and leads to a sustained senescence-associated inflammatory responseSemantic proximity Read ↗

Uncovering a unique pathogenic mechanism of SARS-CoV-2 omicron variant: selective induction of cellular senescenceSemantic proximity Read ↗

Markers of cellular senescence is associated with persistent pulmonary pathology after COVID-19 infectionSemantic proximity Read ↗

The struggle of a good friend getting old: cellular senescence in viral responses and therapy.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.