Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

KRAS(G12D) drives lepidic adenocarcinoma through stem-cell reprogramming

19 Jul 2023

Abstract excerpt

No abstract is available from the source.

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Cancer Cells and Metastasis
Share to Cancer Genomics and Diagnostics
Share to Lung Cancer Treatments and Mutations

Source

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 37468622
DOI: 10.1038/s41586-023-06324-w

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 4 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

KRAS(G12D) drives lepidic adenocarcinoma through stem-cell reprogrammingPMID 37468622Read ↗

Adeno-to-squamous transition drives resistance to KRAS inhibition in <i>LKB1</i> mutant lung cancerSemantic proximity Read ↗

Adeno-to-squamous transition drives resistance to KRAS inhibition in LKB1 mutant lung cancer.Semantic proximity Read ↗

Behind the Wheel of Epithelial Plasticity in KRAS-Driven CancersSemantic proximity Read ↗

Tracing the origins of KRAS oncogene addiction.Semantic proximity Read ↗

Requirement of NEMO/IKKγ for effective expansion of KRAS-induced precancerous lesions in the pancreasSemantic proximity Read ↗

Extrinsic KRAS signaling shapes the pancreatic microenvironment through fibroblast reprogrammingSemantic proximity Read ↗

KRAS mutations take different trajectories to drive pancreatic transformation.Semantic proximity Read ↗

Wnt-deficient and hypoxic environment orchestrates squamous reprogramming of human pancreatic ductal adenocarcinomaSemantic proximity Read ↗

Changes in the Transcriptome and Synthetic Lethal Dependencies Following <i>KRAS</i> Mutant Expression Reveal Profound Tissue-SpecificitySemantic proximity Read ↗

An <i>in vivo</i> KRAS allelic series reveals distinct phenotypes of common oncogenic variantsSemantic proximity Read ↗

Long Noncoding RNAs Preserve Pancreatic Cancer Identity and Resist Cell Fate ConversionSemantic proximity Read ↗

Targeting KRAS: Good, But Is It Good Enough?Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.