Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Genetic variation and elevated liver enzymes during childhood, adolescence and early adulthood.

International journal of epidemiology - 5 Oct 2023

Stender Stefan, Davey Smith George, Richardson Tom G

Abstract excerpt

BACKGROUND: Genetic factors influence the risk of fatty liver disease (FLD) in adults. The aim of this study was to test if, and when, genetic risk factors known to affect FLD in adults begin to exert their deleterious effects during childhood, adolescence and early adulthood. METHODS: We included up to 4018 British children and adolescents from the Avon Longitudinal Study of Parents and Children (ALSPAC) cohort....

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Adolescent
Share to Alanine Transaminase
Share to Aspartate Aminotransferases

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Fibrosis

Share to Longitudinal Studies

Share to Non-alcoholic Fatty Liver Disease

Share to Polymorphism, Single Nucleotide

Source

Open in PubMed Open full text

Identifiers

PMID: 37105232
DOI: 10.1093/ije/dyad048
PMCID: PMC10555681

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 4 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Genetic variation and elevated liver enzymes during childhood, adolescence and early adulthood.PMID 37105232Read ↗

Update on NAFLD genetics: From new variants to the clinic.Semantic proximity Read ↗

High inherited risk predicts age-associated increases in fibrosis in patients with MASLD.Semantic proximity Read ↗

The contribution of genetics and epigenetics to MAFLD susceptibility.Semantic proximity Read ↗

Genetic risk amplifies lifestyle effects on hepatic steatosis and its progression: Insights from a population-based cohort.Semantic proximity Read ↗

Genetic Risk Factors for Metabolic Dysfunction-Associated Steatotic Liver Disease.Semantic proximity Read ↗

Leveraging Human Genetics to Identify Potential New Treatments for Fatty Liver Disease.Semantic proximity Read ↗

Childhood predictors of adult fatty liver. The Cardiovascular Risk in Young Finns Study.Semantic proximity Read ↗

Genetic modulators of metabolic dysfunction-associated steatotic liver disease (MASLD) and their epistatic interactions: from in vitro and animal models to clinical outcomes.Semantic proximity Read ↗

Advances in the genetics of nonalcoholic fatty liver disease.Semantic proximity Read ↗

The impact of genetic and environmental risk factors on adverse liver outcomes.Semantic proximity Read ↗

A Polygenic Risk Score to Refine Risk Stratification and Prediction for Severe Liver Disease by Clinical Fibrosis ScoresSemantic proximity Read ↗

Genomic Signatures of MASLD: How Genomics Is Redefining Our Understanding of Metabolic Liver DiseaseSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.