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Article

FOXA1 in prostate cancer.

Asian journal of andrology - 1 Jan 2000

Dong Hui-Yu, Ding Lei, Zhou Tian-Ren, Yan Tao, Li Jie, Liang Chao

Abstract excerpt

Most prostate cancers initially respond to androgen deprivation therapy (ADT). With the long-term application of ADT, localized prostate cancer will progress to castration-resistant prostate cancer (CRPC), metastatic CRPC (mCRPC), and neuroendocrine prostate cancer (NEPC), and the transcriptional network shifted. Forkhead box protein A1 (FOXA1) may play a key role in this process through multiple mechanisms. To...

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Share to Androgen Antagonists
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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

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Share to Prostatic Neoplasms, Castration-Resistant

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Identifiers

PMID: 36018068
DOI: 10.4103/aja202259
PMCID: PMC10226509

Provenance

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FOXA1 in prostate cancer.PMID 36018068Read ↗

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Elevated levels of FOXA1 facilitate androgen receptor chromatin binding resulting in a CRPC-like phenotype.Semantic proximity Read ↗

Androgen receptor-independent function of FoxA1 in prostate cancer metastasis.Semantic proximity Read ↗

SETD1A Promotes Proliferation of Castration-Resistant Prostate Cancer Cells via FOXM1 TranscriptionSemantic proximity Read ↗

Noncoding mutations target cis-regulatory elements of the FOXA1 plexus in prostate cancer.Semantic proximity Read ↗

FOXM1 promotes the progression of prostate cancer by regulating PSA gene transcriptionSemantic proximity Read ↗

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