Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Identification of differentially expressed genes and pathways in diquat and paraquat poisoning using bioinformatics analysis

8 Apr 2022

Abstract excerpt

Objective In this study, differentially expressed genes (DEGs) and signaling pathways involved in diquat (DQ) and paraquat (PQ) poisoning were identified via bioinformatics analysis, in order to inform the development of novel clinical treatments.Methods Raw data from GSE153959 were downloaded from the Gene Expression Omnibus database. DEGs of the DQ vs. control (CON) and PQ vs. CON comparison groups were...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Peroxisome Proliferator-Activated Receptors
Share to Pesticide Exposure and Toxicity

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 35392760
DOI: 10.1080/15376516.2022.2063095

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Identification of differentially expressed genes and pathways in diquat and paraquat poisoning using bioinformatics analysisPMID 35392760Read ↗

Identification of Hub Genes and Key Pathways of Paraquat Induced Human Embryonic Pulmonary Fibrosis by Bioinformatics Analysis and Vitro ExperimentsSemantic proximity Read ↗

Combined Signaling of NF-kappaB and IL-17 Contributes to Mesenchymal Stem Cells-Mediated Protection for Paraquat-induced Acute Lung InjurySemantic proximity Read ↗

The transcription factor, Nuclear factor, erythroid 2 (Nfe2), is a regulator of the oxidative stress response during Danio rerio development.Semantic proximity Read ↗

Time-resolved proteome and transcriptome of paraquat-induced pulmonary fibrosisSemantic proximity Read ↗

Gene expression analysis of the lung following paraquat administration in rats using DNA microarray.Semantic proximity Read ↗

Network pharmacology and molecular docking to elucidate the common mechanism of hydroxychloroquine treatment in lupus nephritis and IgA nephropathySemantic proximity Read ↗

Chromatin Accessibility and Transcriptomic Alterations in Murine Ovarian Granulosa Cells upon Deoxynivalenol ExposureSemantic proximity Read ↗

Identification of differentially expressed genes for fescue toxicosis in high and low tolerant Angus cowsSemantic proximity Read ↗

Identification of the Hub Genes Linked to Lead (IV)-Induced Spleen Toxicity Using the Rat ModelSemantic proximity Read ↗

Acute oxidative stress is associated with cell proliferation in the mouse liverSemantic proximity Read ↗

Oxidative metabolic pathway of diquat mediated by aldehyde oxidase and its detoxification effect in vitroSemantic proximity Read ↗

Use of traditional end points and gene dysregulation to understand mechanisms of toxicity: toxicogenomics in mechanistic toxicology.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.