Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Weak UVB Irradiation Promotes Macrophage M2 Polarization and Stabilizes Atherosclerosis.

Journal of cardiovascular translational research - 1 Aug 2022

Li Xin-Yun, Qin Tao, Zhang Peng-Fei, Yan Wen-Jiang, Lei Ling-Li, Kuang Jiang-Ying, Li Hao-Dong, Zhang Wen-Cheng, Lu Xiao-Ting, Sun Yuan-Yuan

Abstract excerpt

Atherosclerosis (AS) is a chronic cardiovascular disease endangering human health and is one of the most common causes of myocardial infarction and stroke. Macrophage polarization plays a vital role in regulating plaque stability. As an important component of sunlight, ultraviolet B (UVB) has been proven to promote vitamin D and nitric oxide synthesis. This research used an AS model in ApoE-/- mice to study the...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Atherosclerosis
Share to Macrophage Activation

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Macrophages

Share to Phenotype

Share to Plaque, Atherosclerotic

Source

Open in PubMed Open full text

Identifiers

PMID: 34811697
DOI: 10.1007/s12265-021-10189-7
PMCID: PMC9622510

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Weak UVB Irradiation Promotes Macrophage M2 Polarization and Stabilizes Atherosclerosis.PMID 34811697Read ↗

UVB Exposure Prevents Atherosclerosis by Regulating Immunoinflammatory Responses.Semantic proximity Read ↗

Ginsenoside Rb1 enhances atherosclerotic plaque stability by skewing macrophages to the M2 phenotype.Semantic proximity Read ↗

The role of metabolic reprogramming of oxygen-induced macrophages in the dynamic changes of atherosclerotic plaques.Semantic proximity Read ↗

Salvianolic acid B inhibits RAW264.7 cell polarization towards the M1 phenotype by inhibiting NF-κB and Akt/mTOR pathway activation.Semantic proximity Read ↗

Radiation-Induced Macrophage Senescence Impairs Resolution Programs and Drives Cardiovascular InflammationSemantic proximity Read ↗

Photobiomodulation induces murine macrophages polarization toward M2 phenotype.Semantic proximity Read ↗

β-carotene accelerates the resolution of atherosclerosis in miceSemantic proximity Read ↗

[Zhuyu Pills promote polarization of macrophages toward M2 phenotype to prevent atherosclerosis via PPARγ/NF-κB signaling pathway].Semantic proximity Read ↗

Ablative fractional CO2 laser treatment promotes wound healing phenotype in skin macrophages.Semantic proximity Read ↗

5,7‐Dimethoxyflavone, an activator of PPARα/γ, inhibits UVB‐induced MMP expression in human skin fibroblast cellsSemantic proximity Read ↗

[Correlation between autophagy and polarization of macrophages in atherosclerosis plaque in arteriosclerosis obliterans amputees].Semantic proximity Read ↗

Skin-Derived Mesenchymal Stem Cells Alleviate Atherosclerosis via Modulating Macrophage FunctionSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.