Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

DNA Methylation Patterns in CD4+ T Cells of Naïve and Influenza A Virus-Infected Mice Developmentally Exposed to an Aryl Hydrocarbon Receptor Ligand

1 Jan 2021

Abstract excerpt

Background: Early life environmental exposures can have lasting effects on the function of the immune system and contribute to disease later in life. Epidemiological studies have linked early life exposure to xenobiotics that bind the aryl hydrocarbon receptor (AhR) with dysregulated immune responses later in life. Among the immune cells influenced by developmental activation of the AhR are CD4+ T cells. Yet, the...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Birth, Development, and Health
Share to Diabetes and associated disorders
Share to Epigenetics and DNA Methylation

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 33449811
DOI: 10.1289/ehp7699

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 5 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

DNA Methylation Patterns in CD4+ T Cells of Naïve and Influenza A Virus-Infected Mice Developmentally Exposed to an Aryl Hydrocarbon Receptor LigandPMID 33449811Read ↗

Developmental Activation of the AHR Increases Effector CD4+ T Cells and Exacerbates Symptoms in Autoimmune Disease-Prone Gnaq+/- Mice.Semantic proximity Read ↗

Intersection of Epigenetic and Immune Alterations: Implications for Fetal Alcohol Spectrum Disorder and Mental HealthSemantic proximity Read ↗

Epigenetic underpinnings of developmental immunotoxicity and autoimmune diseaseSemantic proximity Read ↗

Prenatal Exposures to Persistent and Non‐Persistent Organic Compounds and Effects on Immune System DevelopmentSemantic proximity Read ↗

AhR Activation Leads to Attenuation of Murine Autoimmune Hepatitis: Single-Cell RNA-Seq Analysis Reveals Unique Immune Cell Phenotypes and Gene Expression Changes in the Liver.Semantic proximity Read ↗

Epigenetic changes associated with disease progression in a mouse model of childhood allergic asthmaSemantic proximity Read ↗

The managed immune system: protecting the womb to delay the tombSemantic proximity Read ↗

Early life exposure to cigarette smoke primes lung function and DNA methylation changes at <i>Cyp1a1</i> upon exposure later in lifeSemantic proximity Read ↗

Early-life Exposure to Arsenic Primes the Offspring to Increased Asthma Risk: Transcriptome and Epigenome AnalysisSemantic proximity Read ↗

Perinatal exposure to lead results in altered DNA methylation in adult mouse liver and blood: Implications for target versus surrogate tissue use in environmental epigeneticsSemantic proximity Read ↗

Prenatal TCDD causes persistent modulation of the postnatal immune response, and exacerbates inflammatory disease, in 36-week-old lupus-like autoimmune SNF1 mice.Semantic proximity Read ↗

The immune phenotype of AhR null mouse mutants: not a simple mirror of xenobiotic receptor over-activation.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.