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Article

miR-30a regulates γ-globin expression in erythoid precursors of intermedia thalassemia through targeting BCL11A

1 May 2020

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Share to Hemoglobinopathies and Related Disorders
Share to MicroRNA in disease regulation
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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 32406020
DOI: 10.1007/s11033-020-05483-7

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miR-30a regulates γ-globin expression in erythoid precursors of intermedia thalassemia through targeting BCL11APMID 32406020Read ↗

The role of miR-129-5p in regulating γ-globin expression and erythropoiesis in β-thalassemiaSemantic proximity Read ↗

Exploring the crosstalk between long non-coding RNAs and microRNAs to unravel potential prognostic and therapeutic biomarkers in β-thalassemiaSemantic proximity Read ↗

Upregulation of miR-17-3p is associated with HbF in patients with β-thalassemia and induces γ-globin expression by targeting BCL11ASemantic proximity Read ↗

Regulation of miR-17-3p on K562 cell proliferation and erythroid differentiation as well as γ-globin expression in patients with β-thalassemiarSemantic proximity Read ↗

MicroRNA-486-3p regulates γ-globin expression in human erythroid cells by directly modulating BCL11A.Semantic proximity Read ↗

Upregulation of miR‑6747‑3p affects red blood cell lineage development and induces fetal hemoglobin expression by targeting BCL11A in β‑thalassemiaSemantic proximity Read ↗

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Expression of microRNA-451 in normal and thalassemic erythropoiesisSemantic proximity Read ↗

BCL11A mRNA Targeting by miR-210: A Possible Network Regulating γ-Globin Gene ExpressionSemantic proximity Read ↗

MicroRNA expression patterns in HbE/β-thalassemia patients: The passwords to unlock fetal hemoglobin expression in β-hemoglobinopathiesSemantic proximity Read ↗

BCL11A-deficient human erythropoiesis is impaired in vitro and after xenotransplantation into miceSemantic proximity Read ↗

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