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Article

Polymorphisms rs763110 in FASL is linked to hepatitis C virus infection among high-risk populations.

British journal of biomedical science - 1 Jul 2020

Huang P, Wang C H, Zhuo L Y, Xia X S, Yang S, Zhang J W, Fan H Z, Wu J J, Yu R, Yue M, Zhang Y

Abstract excerpt

BACKGROUND: The Fas cell surface death receptor (FAS) and Fas ligand (FASL) can participate in the apoptosis of immune cells and target cells infected with a virus through the FAS-FASL signalling pathway. The decoy receptor 3 (DCR3) can competitively inhibit the binding of FAS to FASL. Our aim is to investigate the effect of single nucleotide polymorphisms (SNPs) in FAS, FASL and DCR3 on hepatitis C virus (HCV)...

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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

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Identifiers

PMID: 32209020
DOI: 10.1080/09674845.2020.1747182

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PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

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