Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

A Phenome-Wide Mendelian Randomization Study of Pancreatic Cancer Using Summary Genetic Data

17 Jul 2019

Abstract excerpt

Abstract Background: The 5-year mortality rate for pancreatic cancer is among the highest of all cancers. Greater understanding of underlying causes could inform population-wide intervention strategies for prevention. Summary genetic data from genome-wide association studies (GWAS) have become available for thousands of phenotypes. These data can be exploited in Mendelian randomization (MR) phenome-wide...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Genetic Associations and Epidemiology
Share to Pancreatic and Hepatic Oncology Research
Share to Pancreatitis Pathology and Treatment

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 31315910
DOI: 10.1158/1055-9965.epi-19-0036

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 4 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

A Phenome-Wide Mendelian Randomization Study of Pancreatic Cancer Using Summary Genetic DataPMID 31315910Read ↗

Exploring Potential Drug Targets for Pancreatic Cancer Based on Mendelian RandomizationSemantic proximity Read ↗

A MR-PheWAS and bidirectional Mendelian randomization study: Exploring for causal relationships of pancreatic cancer.Semantic proximity Read ↗

Integrating plasma protein-centric multi-omics to identify potential therapeutic targets for pancreatic cancer.Semantic proximity Read ↗

Mendelian randomisation study of the effects of known and putative risk factors on pancreatic cancer.Semantic proximity Read ↗

Plasma metabolites as mediators in immune cell-pancreatic cancer risk: insights from Mendelian randomization.Semantic proximity Read ↗

Evidence for a causal link between intra-pancreatic fat deposition and pancreatic cancer: A prospective cohort and Mendelian randomization studySemantic proximity Read ↗

Genetically predicted blood metabolites mediate the association between circulating immune cells and pancreatic cancer: A Mendelian randomization study.Semantic proximity Read ↗

HDL Cholesterol Levels and Pancreatic Cancer Risk: Protective Effects Revealed.Semantic proximity Read ↗

Exploring dietary habits strongly associated with pancreatic cancer from the perspective of Mendelian randomization.Semantic proximity Read ↗

Proteome-Wide Mendelian Randomization Identifies Causal Links Between Blood Proteins and Acute Pancreatitis.Semantic proximity Read ↗

The Role of Obesity, Type 2 Diabetes, and Metabolic Factors in Pancreatic Cancer: A Mendelian Randomization Study.Semantic proximity Read ↗

The exposome and pancreatic cancer, lifestyle and environmental risk factors for PDAC.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.