Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

The Transcription Factor ETV5 Mediates BRAFV600E-Induced Proliferation and TWIST1 Expression in Papillary Thyroid Cancer Cells

25 Sept 2018

Abstract excerpt

The ETS family of transcription factors is involved in several normal remodeling events and pathological processes including tumor progression. ETS transcription factors are divided into subfamilies based on the sequence and location of the ETS domain. ETV5 (Ets variant gene 5; also known as ERM) is a member of the PEA3 subfamily. Our meta-analysis of normal, benign, and malignant thyroid samples demonstrated...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Cancer-related Molecular Pathways
Share to PI3K/AKT/mTOR signaling in cancer
Share to Thyroid Cancer Diagnosis and Treatment

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 30265861
DOI: 10.1016/j.neo.2018.09.003

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 8 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

The Transcription Factor ETV5 Mediates BRAFV600E-Induced Proliferation and TWIST1 Expression in Papillary Thyroid Cancer CellsPMID 30265861Read ↗

Identification of ETV5 as a prognostic marker related to epigenetic modification in pan-cancer and facilitates tumor progression in hepatocellular carcinomaSemantic proximity Read ↗

E26 transformation (ETS)‑specific related transcription factor‑3 (ELF3) orchestrates a positive feedback loop that constitutively activates the MAPK/Erk pathway to drive thyroid cancerSemantic proximity Read ↗

Role of the ETV5/p38 signaling axis in aggressive thyroid cancer cellsSemantic proximity Read ↗

BRAF-induced EHF Expression Affects TERT in Aggressive Papillary Thyroid CancerSemantic proximity Read ↗

Role of BRAF and RAS Mutations in Extrathyroidal Extension in Papillary Thyroid Cancer.Semantic proximity Read ↗

Interaction of BRAF-induced ETS factors with mutant TERT promoter in papillary thyroid cancerSemantic proximity Read ↗

Expression and Clinical Utility of Transcription Factors Involved in Epithelial–Mesenchymal Transition during Thyroid Cancer ProgressionSemantic proximity Read ↗

Overexpression of TPM4 promotes cancer progression through EMT in anaplastic thyroid cancerSemantic proximity Read ↗

The BRAFV600E mutation: what is it really orchestrating in thyroid cancer?Semantic proximity Read ↗

E26 transformation-specific transcription variant 5 in development and cancer: modification, regulation and functionSemantic proximity Read ↗

E26 transformation-specific variant 4 as a tumor promotor in human cancers through specific molecular mechanismsSemantic proximity Read ↗

LEM domain containing 1 promotes thyroid cancer cell proliferation and migration by activating the Wnt/β‑catenin signaling pathway and epithelial‑mesenchymal transitionSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.