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Article

Role of miR-29 in mediating offspring lung phenotype in a rodent model of intrauterine growth restriction.

American journal of physiology. Regulatory, integrative and comparative physiology - 1 Nov 2018

Chuang Tsai-Der, Sakurai Reiko, Gong Ming, Khorram Omid, Rehan Virender K

Abstract excerpt

Considerable epidemiological and experimental evidence supports the concept that the adult chronic lung disease (CLD), is due, at least in part, to aberrations in early lung development in response to an abnormal intrauterine environment; however, the underlying molecular mechanisms remain unknown. We used a well-established rat model of maternal undernutrition (MUN) during pregnancy that results in offspring...

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Share to Extracellular Matrix Proteins
Share to Fetal Growth Retardation

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Glucocorticoids

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Share to Pregnancy

Share to Prenatal Exposure Delayed Effects

Share to Rats, Sprague-Dawley

Source

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Identifiers

PMID: 30088984
DOI: 10.1152/ajpregu.00155.2018
PMCID: PMC6295497

Genes

COL3A1
COL4A5
GC

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

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