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Article

AKT activation is a feature of CALR mutant myeloproliferative neoplasms.

Leukemia - 1 Jan 2019

Fu Chunling, Wen Qiang Jeremy, Marinaccio Christian, Ling Te, Chen Wei, Bulic Marinka, Lasho Terra, Tefferi Ayalew, Crispino John D, Xu Kailin

Abstract excerpt

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Topics

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Share to Calreticulin
Share to Cells, Cultured
Share to CALR gene
Share to Hematopoietic Stem Cells

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Mutation

Share to Myeloproliferative Disorders

Share to Proto-Oncogene Proteins c-akt

Source

Open in PubMed Open full text

Identifiers

PMID: 30082822
DOI: 10.1038/s41375-018-0224-8
PMCID: PMC6328327

Genes

CALR

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026.

Related research

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Research trail→

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AKT activation is a feature of CALR mutant myeloproliferative neoplasms.PMID 30082822Read ↗

JAK Inhibitor in CALR-Mutant MyelofibrosisSemantic proximity Read ↗

Myeloproliferative neoplasms with a low (<5%) CALR mutation allele burden.Semantic proximity Read ↗

Acquisition of JAK2 V617F to CALR-mutated clones accelerates disease progression and might enhance growth capacity.Semantic proximity Read ↗

A CALR mutation preceding BCR-ABL1 in an atypical myeloproliferative neoplasm.Semantic proximity Read ↗

The CALR exon 9 mutations are shared neoantigens in patients with CALR mutant chronic myeloproliferative neoplasmsSemantic proximity Read ↗

A report on the co-occurrence of JAK2V617F and CALR mutations in myeloproliferative neoplasm patients.Semantic proximity Read ↗

The Role of Mutated Calreticulin in the Pathogenesis of BCR-ABL1-Negative Myeloproliferative Neoplasms.Semantic proximity Read ↗

Co-operating STAT5 and AKT signaling pathways in chronic myeloid leukemia and mastocytosis: possible new targets of therapy.Semantic proximity Read ↗

CALR-mutated primary myelofibrosis evolving to chronic myeloid leukemia with both CALR mutation and BCR-ABL1 fusion gene.Semantic proximity Read ↗

Thrombopoietin receptor is required for the oncogenic function of CALR mutantsSemantic proximity Read ↗

Expression of CALR mutants causes mpl-dependent thrombocytosis in zebrafish.Semantic proximity Read ↗

Looking for CALR mutations in familial myeloproliferative neoplasmsSemantic proximity Read ↗

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