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Article

The CALR exon 9 mutations are shared neoantigens in patients with CALR mutant chronic myeloproliferative neoplasms

18 Aug 2016

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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 27560107
DOI: 10.1038/leu.2016.233

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Related research

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Research trail→

Click a neighbor to travelStep 1 · 12 closest

The CALR exon 9 mutations are shared neoantigens in patients with CALR mutant chronic myeloproliferative neoplasmsPMID 27560107Read ↗

Myeloproliferative neoplasms with a low (<5%) CALR mutation allele burden.Semantic proximity Read ↗

Looking for CALR mutations in familial myeloproliferative neoplasmsSemantic proximity Read ↗

Hematopoietic expression of a chimeric murine‐human CALR oncoprotein allows the assessment of anti‐CALR antibody immunotherapies in vivoSemantic proximity Read ↗

A report on the co-occurrence of JAK2V617F and CALR mutations in myeloproliferative neoplasm patients.Semantic proximity Read ↗

Calreticulin exon 9 mutations in myeloproliferative neoplasms.Semantic proximity Read ↗

From JAK to CALR: redefining therapeutic targets in myeloproliferative neoplasms.Semantic proximity Read ↗

[New disease markers within the chronic myeloproliferative neoplasms].Semantic proximity Read ↗

Novel strategies targeting mutant calreticulin in essential thrombocythemia and myelofibrosis.Semantic proximity Read ↗

CALR-mutated primary myelofibrosis evolving to chronic myeloid leukemia with both CALR mutation and BCR-ABL1 fusion gene.Semantic proximity Read ↗

AKT activation is a feature of CALR mutant myeloproliferative neoplasms.Semantic proximity Read ↗

Thrombopoietin receptor is required for the oncogenic function of CALR mutantsSemantic proximity Read ↗

Therapeutic Cancer Vaccination With a Peptide Derived From the Calreticulin Exon 9 Mutations Induces Strong Cellular Immune Responses in Patients With CALR-Mutant Chronic Myeloproliferative NeoplasmsSemantic proximity Read ↗

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