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Article

Acquisition of JAK2 V617F to CALR-mutated clones accelerates disease progression and might enhance growth capacity.

British journal of haematology - 1 Sept 2021

Nishimura Misa, Nagaharu Keiki, Ikejiri Makoto, Sugimoto Yuka, Sasao Ryota, Ohya Eiko, Mizutani Minoru, Ohishi Kohshi, Tawara Isao, Sekine Takao

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PMID: 34060068
DOI: 10.1111/bjh.17571

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Related research

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Acquisition of JAK2 V617F to CALR-mutated clones accelerates disease progression and might enhance growth capacity.PMID 34060068Read ↗

JAK2 V617F and CALR mutations are not mutually exclusive; findings from retrospective analysis of a small patient cohort.Semantic proximity Read ↗

JAK2V617F and CALR double mutations are more frequently encountered in patients with low JAK2V617F allelic burdens.Semantic proximity Read ↗

From JAK to CALR: redefining therapeutic targets in myeloproliferative neoplasms.Semantic proximity Read ↗

A report on the co-occurrence of JAK2V617F and CALR mutations in myeloproliferative neoplasm patients.Semantic proximity Read ↗

BCR-ABL1-positive and JAK2 V617F-positive clones in 23 patients with both aberrations reveal biologic and clinical importance.Semantic proximity Read ↗

JAK Inhibitor in CALR-Mutant MyelofibrosisSemantic proximity Read ↗

Co-occurrence of JAK2 V617F and an uncommon CALR del (p.K368fs*51) mutation facilitates JAK2/STAT signaling in polycythemia vera.Semantic proximity Read ↗

[Latest Research Advance of Myeloproliferative Diseases Related Genes-Review].Semantic proximity Read ↗

Increased B cell activation is present in JAK2V617F-mutated, CALR-mutated and triple-negative essential thrombocythemia.Semantic proximity Read ↗

Clinical effect of driver mutations of JAK2, CALR, or MPL in primary myelofibrosis.Semantic proximity Read ↗

Myeloproliferative neoplasms with a low (<5%) CALR mutation allele burden.Semantic proximity Read ↗

JAK2 V617F clonal disorders: fate or chance?Semantic proximity Read ↗

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