Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Decreased Siglec-9 Expression on Natural Killer Cell Subset Associated With Persistent HBV Replication.

Frontiers in immunology - 1 Jan 2018

Zhao Di, Jiang Xuemei, Xu Yong, Yang Huimin, Gao Dongni, Li Xueen, Gao Lifen, Ma Chunhong, Liang Xiaohong

Abstract excerpt

Siglec-9 is an MHC-independent inhibitory receptor selectively expressed on CD56dim NK cells. Its role in infection diseases has not been investigated yet. Here, we studied the potential regulatory roles of NK Siglec-9 in the pathogenesis of chronic hepatitis B (CHB) infection. Flow cytometry evaluated the expression of Siglec-9 and other receptors on peripheral NK cells. Immunofluorescence staining was used to...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Biopsy
Share to Fluorescent Antibody Technique

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Hepatitis B

Share to Hepatitis B virus

Share to Hepatocytes

Share to Immunophenotyping

Share to Killer Cells, Natural

Share to Leukocytes, Mononuclear

Source

Open in PubMed Open full text

Identifiers

PMID: 29899741
DOI: 10.3389/fimmu.2018.01124
PMCID: PMC5988867

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Decreased Siglec-9 Expression on Natural Killer Cell Subset Associated With Persistent HBV Replication.PMID 29899741Read ↗

NK cells in hepatitis B virus infection: a potent target for immunotherapy.Semantic proximity Read ↗

Lack of Siglec-7 expression identifies a dysfunctional natural killer cell subset associated with liver inflammation and fibrosis in chronic HCV infection.Semantic proximity Read ↗

The expression of TLR-9, CD86, and CD95 phenotypes in circulating B cells of patients with chronic viral hepatitis B or C before and after antiviral therapy.Semantic proximity Read ↗

Peripheral NK cell phenotypic alteration and dysfunctional state post hepatitis B subviral particles stimulation in CHB patients: evading immune surveillance.Semantic proximity Read ↗

NK cell phenotypic and functional shifts coincide with specific clinical phases in the natural history of chronic HBV infection.Semantic proximity Read ↗

Human intrahepatic CD69 + CD8+ T cells have a tissue resident memory T cell phenotype with reduced cytolytic capacity.Semantic proximity Read ↗

CMV drives clonal expansion of NKG2C + NK cells expressing self‐specific KIRs in chronic hepatitis patientsSemantic proximity Read ↗

Natural killer (NK) and HLA-C in in hepatitis C virus infected patient in Egypt during infection with Covid 19Semantic proximity Read ↗

Compromised function of natural killer cells in acute and chronic viral hepatitis.Semantic proximity Read ↗

Natural killer cell function and dysfunction in hepatitis C virus infection.Semantic proximity Read ↗

Differential alteration of CD56(bright) and CD56 (dim) natural killer cells in frequency, phenotype, and cytokine response in chronic hepatitis C virus infection.Semantic proximity Read ↗

Phenotypic characteristics of natural killer cells in acute hepatitis.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.