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Article

Direct Promoter Repression by BCL11A Controls the Fetal to Adult Hemoglobin Switch

29 Mar 2018

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Share to Blood groups and transfusion
Share to Hemoglobinopathies and Related Disorders
Share to Prenatal Screening and Diagnostics

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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 29606353
DOI: 10.1016/j.cell.2018.03.016

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Related research

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Research trail→

Click a neighbor to travelStep 1 · 12 closest

Direct Promoter Repression by BCL11A Controls the Fetal to Adult Hemoglobin SwitchPMID 29606353Read ↗

Hemoglobin switching's surprise: the versatile transcription factor BCL11A is a master repressor of fetal hemoglobinSemantic proximity Read ↗

Update on fetal hemoglobin gene regulation in hemoglobinopathiesSemantic proximity Read ↗

Transcriptional silencing of fetal hemoglobin by BCL11ASemantic proximity Read ↗

Regulation of the fetal hemoglobin silencing factor BCL11ASemantic proximity Read ↗

Human fetal hemoglobin expression is regulated by the developmental stage-specific repressor BCL11A.Semantic proximity Read ↗

The Fetal-to-Adult Hemoglobin Switch — Mechanism and TherapySemantic proximity Read ↗

Molecular analysis of the erythroid phenotype of a patient with BCL11A haploinsufficiency.Semantic proximity Read ↗

Human Fetal Hemoglobin Expression Is Regulated by the Developmental Stage-Specific Repressor BCL11ASemantic proximity Read ↗

Corepressor-dependent silencing of fetal hemoglobin expression by BCL11ASemantic proximity Read ↗

Transcription factors LRF and BCL11A independently repress expression of fetal hemoglobinSemantic proximity Read ↗

Targeted Therapeutic Strategies for Fetal Hemoglobin InductionSemantic proximity Read ↗

Control of human hemoglobin switching by LIN28B-mediated regulation of BCL11A translationSemantic proximity Read ↗

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