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Article

Hemoglobin switching's surprise: the versatile transcription factor BCL11A is a master repressor of fetal hemoglobin

1 Aug 2015

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Share to Epigenetics and DNA Methylation
Share to Hemoglobinopathies and Related Disorders
Share to Prenatal Screening and Diagnostics

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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 26375765
DOI: 10.1016/j.gde.2015.08.001

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PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 6 Sept 2026.

Related research

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Research trail→

Click a neighbor to travelStep 1 · 12 closest

Hemoglobin switching's surprise: the versatile transcription factor BCL11A is a master repressor of fetal hemoglobinPMID 26375765Read ↗

Direct Promoter Repression by BCL11A Controls the Fetal to Adult Hemoglobin SwitchSemantic proximity Read ↗

The Fetal-to-Adult Hemoglobin Switch — Mechanism and TherapySemantic proximity Read ↗

Regulation of the fetal hemoglobin silencing factor BCL11ASemantic proximity Read ↗

Update on fetal hemoglobin gene regulation in hemoglobinopathiesSemantic proximity Read ↗

Human fetal hemoglobin expression is regulated by the developmental stage-specific repressor BCL11A.Semantic proximity Read ↗

Transcriptional silencing of fetal hemoglobin by BCL11ASemantic proximity Read ↗

Corepressor-dependent silencing of fetal hemoglobin expression by BCL11ASemantic proximity Read ↗

Human Fetal Hemoglobin Expression Is Regulated by the Developmental Stage-Specific Repressor BCL11ASemantic proximity Read ↗

Insight of fetal to adult hemoglobin switch: Genetic modulators and therapeutic targetsSemantic proximity Read ↗

Molecular analysis of the erythroid phenotype of a patient with BCL11A haploinsufficiency.Semantic proximity Read ↗

Control of human hemoglobin switching by LIN28B-mediated regulation of BCL11A translationSemantic proximity Read ↗

KLF1 regulates BCL11A expression and γ- to β-globin gene switchingSemantic proximity Read ↗

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