Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Bach2 repression mediates Th17 cell induced inflammation and associates with clinical features of advanced disease in chronic pancreatitis

21 Jun 2017

Abstract excerpt

Objectives Altered immune homeostasis and involvement of T cells has been reported in chronic pancreatitis (CP). We evaluated the role of Bach2 (BTB and CNC homology basic leucine zipper transcription factor 2), a key regulator of immune homeostasis in the chronicity of CP. Methods Expression of Bach2 and T‐cell transcription factors, enumeration of BACH2+/CD4+ T‐lymphocytes were performed by qRT‐PCR and flow...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to IL-33, ST2, and ILC Pathways
Share to Immune Cell Function and Interaction
Share to Pancreatitis Pathology and Treatment

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 29511557
DOI: 10.1177/2050640617716596

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Bach2 repression mediates Th17 cell induced inflammation and associates with clinical features of advanced disease in chronic pancreatitisPMID 29511557Read ↗

Genome-wide Association Analysis for Organ Failure in Acute Pancreatitis.Semantic proximity Read ↗

Pathophysiological role of BACH transcription factors in digestive system diseasesSemantic proximity Read ↗

Single-cell sequencing unveils distinct immune microenvironment with CCR6-CCL20 crosstalk in human chronic pancreatitisSemantic proximity Read ↗

Genome-wide association study identifies inversion in the CTRB1-CTRB2 locus to modify risk for alcoholic and non-alcoholic chronic pancreatitis.Semantic proximity Read ↗

A multi-omics analysis of pancreatitis: bridging familial genetics and disease progression.Semantic proximity Read ↗

Identifying miRNA-mRNA regulation network of chronic pancreatitis based on the significant functional expressionSemantic proximity Read ↗

The role of Bach2 in regulating CD8 + T cell development and functionSemantic proximity Read ↗

Caspase-10 affects the pathogenesis of primary biliary cholangitis by regulating inflammatory cell deathSemantic proximity Read ↗

Chronic pancreatitis: pathogenesis and molecular aspects.Semantic proximity Read ↗

Chronic Pancreatitis: Pediatric and Adult Cohorts Show Similarities in Disease Progress Despite Different Risk Factors.Semantic proximity Read ↗

Chronic pancreatitis patient-derived organoids reveal new paths to precision therapeuticsSemantic proximity Read ↗

Patient-derived organoids reveal ductal dysfunction and CFTR-modulator responses in chronic pancreatitis.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.