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Article

HDAC11 Inhibits Myoblast Differentiation through Repression of MyoD-Dependent Transcription.

Molecules and cells - 30 Sept 2017

Byun Sang Kyung, An Tae Hyeon, Son Min Jeong, Lee Da Som, Kang Hyun Sup, Lee Eun-Woo, Han Baek Soo, Kim Won Kon, Bae Kwang-Hee, Oh Kyoung-Jin, Lee Sang Chul

Abstract excerpt

Abnormal differentiation of muscle is closely associated with aging (sarcopenia) and diseases such as cancer and type II diabetes. Thus, understanding the mechanisms that regulate muscle differentiation will be useful in the treatment and prevention of these conditions. Protein lysine acetylation and methylation are major post-translational modification mechanisms that regulate key cellular processes. In this...

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Topics

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Share to Acetylation
Share to Cell Differentiation

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Histone Deacetylases

Share to MEF2 Transcription Factors

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Source

Open in PubMed Open full text

Identifiers

PMID: 28927261
DOI: 10.14348/molcells.2017.0116
PMCID: PMC5638774

Genes

HDAC11
MYOG

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 5 Sept 2026. Manifest publication-graph-cohort-2026-09-05.1.

Source receipt: pubmed, retrieved 5 Sept 2026.

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