Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Increased levels of mitochondrial DNA copy number in patients with vitiligo

3 Sept 2017

Abstract excerpt

BACKGROUND: Oxidative stress is known to be involved in the pathogenesis of autoimmune diseases such as vitiligo. Evidence suggests that the human mitochondrial DNA copy number (mtDNAcn) is vulnerable to damage mediated by oxidative stress. The purpose of this study was to examine and compare peripheral blood mtDNAcn and oxidative DNA damage byproducts (8-hydroxy-2-deoxyguanosine; 8-OHdG) in patients with...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to melanin and skin pigmentation
Share to Mitochondrial Function and Pathology
Share to Ocular Oncology and Treatments

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 28866865
DOI: 10.1111/ced.13185

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 8 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Increased levels of mitochondrial DNA copy number in patients with vitiligoPMID 28866865Read ↗

Analysis of oxidative stress status, catalase and catechol-O-methyltransferase polymorphisms in Egyptian vitiligo patients.Semantic proximity Read ↗

Involvement of superoxide dismutase isoenzymes and their genetic variants in progression of and higher susceptibility to vitiligo.Semantic proximity Read ↗

Association of SOD1 gene variants (50 bp Ins/Del, rs4817415, rs2070424, rs1041740, rs17880135) with plasma protein levels in vitiligo patients and their analysis in silico.Semantic proximity Read ↗

The relationship between non-segmental Vitiligo, HLA genotype and oxidative stress.Semantic proximity Read ↗

Evaluation of CAT Variants A-89T, C389T, and C419T in Patients with Vitiligo in the Saudi Population.Semantic proximity Read ↗

Immunometabolism in the pathogenesis of vitiligoSemantic proximity Read ↗

A functional single-nucleotide polymorphism in the catechol-O-methyltransferase gene alter vitiligo risk in a Chinese population.Semantic proximity Read ↗

The association of FOXO3A gene polymorphisms with serum FOXO3A levels and oxidative stress markers in vitiligo patients.Semantic proximity Read ↗

The pathogenesis of vitiligo.Semantic proximity Read ↗

Vitiligo: interplay between oxidative stress and immune system.Semantic proximity Read ↗

Increased level of cathelicidin (LL-37) in vitiligo: Possible pathway independent from vitamin D receptor gene polymorphism.Semantic proximity Read ↗

Molecular Marker in Vitiligo: A ReviewSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.