Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Sexual dimorphism of the pulmonary transcriptome in neonatal hyperoxic lung injury: identification of angiogenesis as a key pathway

17 Aug 2017

Abstract excerpt

Bronchopulmonary dysplasia (BPD) is characterized by impaired alveolar secondary septation and vascular growth. Exposure to high concentrations of oxygen (hyperoxia) contributes to the development of BPD. The male sex is considered an independent risk factor for the development of BPD. The reasons underlying sexually dimorphic outcomes in premature neonates are not known. We hypothesized that sex-specific...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Congenital Diaphragmatic Hernia Studies
Share to Neonatal Respiratory Health Research
Share to Pulmonary Hypertension Research and Treatments

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 28818871
DOI: 10.1152/ajplung.00230.2017

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 2 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Sexual dimorphism of the pulmonary transcriptome in neonatal hyperoxic lung injury: identification of angiogenesis as a key pathwayPMID 28818871Read ↗

MicroRNA-30a as a candidate underlying sex-specific differences in neonatal hyperoxic lung injury: implications for BPDSemantic proximity Read ↗

Remarkable sex-specific differences at single-cell resolution in neonatal hyperoxic lung injurySemantic proximity Read ↗

Remarkable sex-specific differences at Single-Cell Resolution in Neonatal Hyperoxic Lung InjurySemantic proximity Read ↗

Proteomic analysis of sex differences in hyperoxic lung injury in neonatal mice.Semantic proximity Read ↗

Neonatal hyperoxia induces sex-dependent pulmonary cellular and transcriptomic changes in an experimental mouse model of bronchopulmonary dysplasiaSemantic proximity Read ↗

Epigenetic response to hyperoxia in the neonatal lung is sexually dimorphicSemantic proximity Read ↗

Modulation of Recovery from Neonatal Hyperoxic Lung Injury by Sex as a Biological VariableSemantic proximity Read ↗

Neonatal hyperoxia induces activated pulmonary cellular states and sex-dependent transcriptomic changes in a model of experimental bronchopulmonary dysplasiaSemantic proximity Read ↗

Role of sex as a biological variable in neonatal alveolar macrophagesSemantic proximity Read ↗

Sex-specific differences in primary neonatal murine lung fibroblasts exposed to hyperoxia in vitro: implications for bronchopulmonary dysplasiaSemantic proximity Read ↗

Antenatal Hypoxia and Pulmonary Vascular Function and RemodelingSemantic proximity Read ↗

A postnatal human lung developmental atlas reveals windows of genetic vulnerability to chronic lung diseaseSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.