Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Proteomic analysis of sex differences in hyperoxic lung injury in neonatal mice.

International journal of medical sciences - 1 Jan 2020

Cheng Huaiping, Wang Huifang, Wu Chantong, Zhang Yuan, Bao Tianping, Tian Zhaofang

Abstract excerpt

Sex-specific differences in the severity of bronchopulmonary dysplasia (BPD) are due to different susceptibility to hyperoxic lung injury, but the mechanism is unclear. In this study, neonatal male and female mouse pups (C57BL/6J) were exposed to hyperoxia and lung tissues were excised on postnatal day 7 for histological analysis and tandem mass tags proteomic analysis. We found that the lung sections from the...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Animals, Newborn
Share to Bronchopulmonary Dysplasia

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Hyperoxia

Share to Lung Injury

Share to Proteomics

Share to Severity of Illness Index

Share to Sex Factors

Source

Open in PubMed Open full text

Identifiers

PMID: 33029086
DOI: 10.7150/ijms.42073
PMCID: PMC7532490

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Proteomic analysis of sex differences in hyperoxic lung injury in neonatal mice.PMID 33029086Read ↗

Sexual dimorphism of the pulmonary transcriptome in neonatal hyperoxic lung injury: identification of angiogenesis as a key pathwaySemantic proximity Read ↗

Sex-specific differences in primary neonatal murine lung fibroblasts exposed to hyperoxia in vitro: implications for bronchopulmonary dysplasiaSemantic proximity Read ↗

Mouse lung development and NOX1 induction during hyperoxia are developmentally regulated and mitochondrial ROS dependentSemantic proximity Read ↗

Remarkable sex-specific differences at single-cell resolution in neonatal hyperoxic lung injurySemantic proximity Read ↗

Neonatal hyperoxia induces activated pulmonary cellular states and sex-dependent transcriptomic changes in a model of experimental bronchopulmonary dysplasiaSemantic proximity Read ↗

Role of sex as a biological variable in neonatal alveolar macrophagesSemantic proximity Read ↗

Epigenetic response to hyperoxia in the neonatal lung is sexually dimorphicSemantic proximity Read ↗

Remarkable sex-specific differences at Single-Cell Resolution in Neonatal Hyperoxic Lung InjurySemantic proximity Read ↗

Neonatal hyperoxia induces sex-dependent pulmonary cellular and transcriptomic changes in an experimental mouse model of bronchopulmonary dysplasiaSemantic proximity Read ↗

Animal models of bronchopulmonary dysplasia. The term mouse modelsSemantic proximity Read ↗

MicroRNA-30a as a candidate underlying sex-specific differences in neonatal hyperoxic lung injury: implications for BPDSemantic proximity Read ↗

Deficiency of SPP1 alleviates hyperoxia-induced bronchopulmonary dysplasia in neonatal miceSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.