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Article

Sema3A promotes the resolution of cardiac inflammation after myocardial infarction.

Basic research in cardiology - 1 Jul 2017

Rienks Marieke, Carai Paolo, Bitsch Nicole, Schellings Mark, Vanhaverbeke Maarten, Verjans Johan, Cuijpers Ilona, Heymans Stephane, Papageorgiou Anna

Abstract excerpt

Optimal healing after myocardial infarction requires not only the induction of inflammation, but also its timely resolution. In patients, 30 days post myocardial infarction, circulating monocytes have increased expression of Semaphorin3A (Sema3A) as compared to directly after admission. This increased expression coincides with increased expression of Cx3CR1-a marker of non-classical monocytes that are important...

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Share to Myocarditis
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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Macrophage Activation

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Source

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Identifiers

PMID: 28540528
DOI: 10.1007/s00395-017-0630-5
PMCID: PMC5443852

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 5 Sept 2026. Manifest publication-graph-cohort-2026-09-05.1.

Source receipt: pubmed, retrieved 5 Sept 2026.

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