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Article

A double-negative feedback loop between EpCAM and ERK contributes to the regulation of epithelial–mesenchymal transition in cancer

13 Feb 2017

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Source

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 28192403
DOI: 10.1038/onc.2016.504

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A double-negative feedback loop between EpCAM and ERK contributes to the regulation of epithelial–mesenchymal transition in cancerPMID 28192403Read ↗

Understanding the versatile roles and applications of EpCAM in cancers: from bench to bedsideSemantic proximity Read ↗

Revisiting the Roles of Pro-Metastatic EpCAM in CancerSemantic proximity Read ↗

The Emerging Role of EpCAM in Cancer and Stem Cell SignalingSemantic proximity Read ↗

Functional Implications of the Dynamic Regulation of EpCAM during Epithelial-to-Mesenchymal TransitionSemantic proximity Read ↗

ERK2-ZEB1-miR-101-1 axis contributes to epithelial–mesenchymal transition and cell migration in cancerSemantic proximity Read ↗

EpCAM in carcinogenesis: the good, the bad or the uglySemantic proximity Read ↗

Molecular mechanisms of epithelial–mesenchymal transitionSemantic proximity Read ↗

Functions of EpCAM in physiological processes and diseases (Review)Semantic proximity Read ↗

EpCAM and its potential role in tumor-initiating cellsSemantic proximity Read ↗

Shedding light on the EpCAM: An overviewSemantic proximity Read ↗

Cancer as a disease of epithelial–mesenchymal interactions and extracellular matrix regulationSemantic proximity Read ↗

The role of EpCAM in physiology and pathology of the epithelium.Semantic proximity Read ↗

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