Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Human amnion favours tissue repair by inducing the M1-to-M2 switch and enhancing M2 macrophage features.

Journal of tissue engineering and regenerative medicine - 1 Oct 2017

Magatti Marta, Vertua Elsa, De Munari Silvia, Caro Marta, Caruso Maddalena, Silini Antonietta, Delgado Mario, Parolini Ornella

Abstract excerpt

Human amniotic mesenchymal cells (hAMTCs) possess interesting immunomodulatory properties, making them attractive candidates for regenerative medicine applications. Recent in vivo reports argue in favour of an important role for macrophages as targets of hAMTC-mediated suppression of inflammation and the enhancement of tissue repair. However, a comprehensive study of the effects of hAMTCs and their conditioned...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Amnion
Share to Cell Polarity

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Culture Media, Conditioned

Share to Phagocytosis

Share to Prostaglandins

Share to Regeneration

Share to U937 Cells

Share to Wound Healing

Source

Open in PubMed Open full text

Identifiers

PMID: 27396853
DOI: 10.1002/term.2193
PMCID: PMC5697700

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Human amnion favours tissue repair by inducing the M1-to-M2 switch and enhancing M2 macrophage features.PMID 27396853Read ↗

An optimized protocol for human M2 macrophages using M-CSF and IL-4/IL-10/TGF-β yields a dominant immunosuppressive phenotype.Semantic proximity Read ↗

Placental Hofbauer Cell Polarization Resists Inflammatory Cues In VitroSemantic proximity Read ↗

A critical guideline for controlling monocyte-derived macrophages phenotypes.Semantic proximity Read ↗

ACVR1R206H extends inflammatory responses in human induced pluripotent stem cell-derived macrophagesSemantic proximity Read ↗

Macrophages at the Fetal–Maternal Interface Express Markers of Alternative Activation and Are Induced by M-CSF and IL-10Semantic proximity Read ↗

Mesenchymal Stem Cells Direct the Immunological Fate of Macrophages.Semantic proximity Read ↗

M1-like macrophage contributes to chondrogenesis in vitro.Semantic proximity Read ↗

Macrophage plasticity and polarization in tissue repair and remodelling.Semantic proximity Read ↗

A new mesenchymal stem cell (MSC) paradigm: polarization into a pro-inflammatory MSC1 or an Immunosuppressive MSC2 phenotype.Semantic proximity Read ↗

Classic and new mediators for in vitro modelling of human macrophages.Semantic proximity Read ↗

TLR2 stimulation impairs anti-inflammatory activity of M2-like macrophages, generating a chimeric M1/M2 phenotype.Semantic proximity Read ↗

The Secretome of Preconditioned Mesenchymal Stem Cells Drives Polarization and Reprogramming of M2a Macrophages toward an IL-10-Producing Phenotype.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.