Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

HIF-3α1 promotes colorectal tumor cell growth by activation of JAK-STAT3 signaling

9 Feb 2016

Abstract excerpt

// Xiang Xue 1 , Kylie Jungles 1, 2 , Gunseli Onder 1 , Jalal Samhoun 1 , Balázs Győrffy 3 , Karin M. Hardiman 4 1 Department of Molecular and Integrative Physiology, University of Michigan, Ann Arbor, MI, USA 2 Saint Mary’s College, Notre Dame, IN, USA 3 MTA TTK Lendület Cancer Biomarker Research Group, MTA-SE Pediatrics and Nephrology Research Group, Semmelweis University 2nd Department of Pediatrics, Budapest,...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Cancer, Hypoxia, and Metabolism
Share to Cancer-related Molecular Pathways
Share to RNA modifications and cancer

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 26871465
DOI: 10.18632/oncotarget.7272

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 8 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

HIF-3α1 promotes colorectal tumor cell growth by activation of JAK-STAT3 signalingPMID 26871465Read ↗

The ratio of STAT1 to STAT3 expression is a determinant of colorectal cancer growthSemantic proximity Read ↗

Hypoxia-inducible factor 3α1 increases epithelial-to-mesenchymal transition and iron uptake to drive colorectal cancer liver metastasis.Semantic proximity Read ↗

The role of hypoxia-inducible factor-1α on colon cancer progression and metastasisSemantic proximity Read ↗

Signal Transducer and Activator of Transcription 3 - A Promising Target in Colitis-Associated CancerSemantic proximity Read ↗

STAT3 and the STAT3‑regulated inhibitor of apoptosis protein survivin as potential therapeutic targets in colorectal cancer (Review)Semantic proximity Read ↗

STAT3 Is Necessary for Proliferation and Survival in Colon Cancer–Initiating CellsSemantic proximity Read ↗

GRIM‐19 repressed hypoxia‐induced invasion and EMT of colorectal cancer by repressing autophagy through inactivation of STAT3/HIF‐1α signaling axisSemantic proximity Read ↗

The IL-6/JAK/STAT3 pathway: Potential therapeutic strategies in treating colorectal cancerSemantic proximity Read ↗

Factor inhibiting HIF1α (FIH-1) functions as a tumor suppressor in human colorectal cancer by repressing HIF1α pathwaySemantic proximity Read ↗

Association analyses of the JAK/STAT signaling pathway with the progression and prognosis of colon cancerSemantic proximity Read ↗

STAT3 Expression, Molecular Features, Inflammation Patterns, and Prognosis in a Database of 724 Colorectal CancersSemantic proximity Read ↗

CSF3 contributes to the invasive and metastatic phenotypes and associates with poor prognosis in patients with colorectal cancer via NF-κB signaling pathwaySemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.