Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Deletion of PdMit1, a homolog of yeast Csg1, affects growth and Ca(2+) sensitivity of the fungus Penicillium digitatum, but does not alter virulence.

Research in microbiology - 1 Apr 2015

Zhu Congyi, Wang Weili, Wang Mingshuang, Ruan Ruoxin, Sun Xuepeng, He Meixian, Mao Cungui, Li Hongye

Abstract excerpt

GDP-mannose:inositol-phosphorylceramide (MIPC) and its derivatives are important for Ca(2+) sensitization of Saccharomyces cerevisiae and for the virulence of Candida albicans, but its role in the virulence of plant fungal pathogens remains unclear. In this study, we report the identification and functional characterization of PdMit1, the gene encoding MIPC synthase in Penicillium digitatum, one of the most...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Calcium Chloride
Share to Citrus

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Fungal Proteins

Share to Glycosphingolipids

Share to Mycelium

Share to Penicillium

Share to Sequence Deletion

Share to Spores, Fungal

Source

Open in PubMed Open full text

Identifiers

PMID: 25725383
DOI: 10.1016/j.resmic.2015.02.001
PMCID: PMC4393796

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Deletion of PdMit1, a homolog of yeast Csg1, affects growth and Ca(2+) sensitivity of the fungus Penicillium digitatum, but does not alter virulence.PMID 25725383Read ↗

Role of TFP1 in vacuolar acidification, oxidative stress and filamentous development in Candida albicans.Semantic proximity Read ↗

Glucosylceramides are required for mycelial growth and full virulence in Penicillium digitatum.Semantic proximity Read ↗

Updating Insights into the Regulatory Mechanisms of Calcineurin-Activated Transcription Factor Crz1 in Pathogenic FungiSemantic proximity Read ↗

Functions of CaPhm7 in the regulation of ion homeostasis, drug tolerance, filamentation and virulence in Candida albicansSemantic proximity Read ↗

Adenylyl cyclase is required for cAMP production, growth, conidial germination, and virulence in the citrus green mold pathogen Penicillium digitatum.Semantic proximity Read ↗

bcpmr1 encodes a P-type Ca(2+)/Mn(2+)-ATPase mediating cell-wall integrity and virulence in the phytopathogen Botrytis cinerea.Semantic proximity Read ↗

The mitA gene of Aspergillus fumigatus is required for mannosylation of inositol-phosphorylceramide, but is dispensable for pathogenicity.Semantic proximity Read ↗

Tfp1 is required for ion homeostasis, fluconazole resistance and N-Acetylglucosamine utilization in Candida albicans.Semantic proximity Read ↗

The myosin motor domain-containing chitin synthase PdChsVII is required for development, cell wall integrity and virulence in the citrus postharvest pathogen Penicillium digitatum.Semantic proximity Read ↗

Functional Genomic Analysis of Fluconazole Susceptibility in the Pathogenic Yeast <i>Candida glabrata</i> : Roles of Calcium Signaling and MitochondriaSemantic proximity Read ↗

SUR1 (CSG1/BCL21), a gene necessary for growth of Saccharomyces cerevisiae in the presence of high Ca2+ concentrations at 37 degrees C, is required for mannosylation of inositolphosphorylceramide.Semantic proximity Read ↗

Relevance of the transcription factor PdSte12 in Penicillium digitatum conidiation and virulence during citrus fruit infection.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.