Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Toxin Synthesis by Clostridium difficile Is Regulated through Quorum Signaling

26 Feb 2015

Abstract excerpt

UNLABELLED: Clostridium difficile infection (CDI) is dramatically increasing as a cause of antibiotic- and hospital-associated diarrhea worldwide. C. difficile, a multidrug-resistant pathogen, flourishes in the colon after the gut microbiota has been altered by antibiotic therapy. Consequently, it produces toxins A and B that directly cause disease. Despite the enormous public health problem posed by this...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Bacterial biofilms and quorum sensing
Share to Clostridium difficile and Clostridium perfringens research
Share to Nosocomial Infections in ICU

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 25714717
DOI: 10.1128/mbio.02569-14

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Toxin Synthesis by Clostridium difficile Is Regulated through Quorum SignalingPMID 25714717Read ↗

The Regulatory Networks That Control Clostridium difficile Toxin SynthesisSemantic proximity Read ↗

<i>Clostridioides difficile</i> exploits toxin-mediated inflammation to alter the host nutritional landscape and exclude competitors from the gut microbiotaSemantic proximity Read ↗

Accessory Gene Regulator-1 Locus Is Essential for Virulence and Pathogenesis of Clostridium difficileSemantic proximity Read ↗

Cyclic diguanylate differentially regulates the expression of virulence factors and pathogenesis-related phenotypes in Clostridioides difficile.Semantic proximity Read ↗

Pathogenicity and virulence of Clostridioides difficileSemantic proximity Read ↗

Clostridium difficile infectionSemantic proximity Read ↗

Clostridioides difficile phage biology and applicationSemantic proximity Read ↗

An Update on Clostridioides difficile Binary ToxinSemantic proximity Read ↗

EndD mediates butyrate-dependent toxin release in <i>Clostridioides difficile</i>Semantic proximity Read ↗

Regulation of toxin production in the pathogenic clostridiaSemantic proximity Read ↗

Bacteriophages Contribute to Shaping Clostridioides (Clostridium) difficile SpeciesSemantic proximity Read ↗

Clostridium difficile Toxins A and B: Insights into Pathogenic Properties and Extraintestinal EffectsSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.