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Article

Dynamics of Lewis b binding and sequence variation of the babA adhesin gene during chronic Helicobacter pylori infection in humans.

mBio - 16 Dec 2014

Nell Sandra, Kennemann Lynn, Schwarz Sandra, Josenhans Christine, Suerbaum Sebastian

Abstract excerpt

UNLABELLED: Helicobacter pylori undergoes rapid microevolution during chronic infection, but very little is known about how this affects host interaction factors. The best-studied adhesin of H. pylori is BabA, which mediates binding to the blood group antigen Lewis b [Le(b)]. To study the dynamics of Le(b) adherence during human infection, we analyzed paired H. pylori isolates obtained sequentially from...

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Share to Adhesins, Bacterial
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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to DNA, Bacterial

Share to Helicobacter Infections

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Source

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Identifiers

PMID: 25516619
DOI: 10.1128/mBio.02281-14
PMCID: PMC4271554

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Dynamics of Lewis b binding and sequence variation of the babA adhesin gene during chronic Helicobacter pylori infection in humans.PMID 25516619Read ↗

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