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Article

Prenatal exposure to cigarette smoke interacts with OPRM1 to modulate dietary preference for fat.

Journal of psychiatry & neuroscience : JPN - 1 Jan 2015

Lee Ken W K, Abrahamowicz Michal, Leonard Gabriel T, Richer Louis, Perron Michel, Veillette Suzanne, Reischl Eva, Bouchard Luigi, Gaudet Daniel, Paus Tomas, Pausova Zdenka

Abstract excerpt

BACKGROUND: Preference for fatty foods is a risk factor for obesity. It is a complex behaviour that involves the brain reward system and is regulated by genetic and environmental factors, such as the opioid receptor mu-1 gene (OPRM1) and prenatal exposure to maternal cigarette smoking (PEMCS). We examined whether OPRM1 and PEMCS interact in influencing fat intake and whether exposure-associated epigenetic...

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Source

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Identifiers

PMID: 25266401
DOI: 10.1503/jpn.130263
PMCID: PMC4275330

Genes

OPRM1

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

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